Xin Qi

Professor

Case Western Reserve University

United States

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About

Professor Xin Qi is currently a faculty member at Case Western Reserve University in the United States. His research primarily focuses on Alzheimer's disease, mitochondrial dysfunction, and neurodegeneration. Recent articles by Professor Qi explore associations between semaglutide and Alzheimer's-related dementias, as well as targeted therapies for Alzheimer's and other neurodegenerative disorders. He aims to advance understanding of mitochondrial dynamics and their implications for conditions such as Parkinson's disease and Huntington's disease.

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UNITED STATES

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Sunil Jamuna Tripathi

Johns Hopkins University

UNITED STATES

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Xin Qi

Research Interests

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Articles (20)

Associations of semaglutide with Alzheimer's disease-related dementias in patients with type 2 diabetes: A real-world target trial emulation study

Mitochondrial dysfunction in Alzheimer's disease: Guiding the path to targeted therapies

Methionine Sulfoxide Speciation in Mouse Hippocampus Revealed by Global Proteomics Exhibits Age- and Alzheimer’s Disease-Dependent Changes Targeted to Mitochondrial and Glycolytic Pathways

Acutely blocking excessive mitochondrial fission prevents chronic neurodegeneration after traumatic brain injury.

Allosteric control of dynamin-related protein 1 through a disordered C-terminal Short Linear Motif

BCKDK loss impairs mitochondrial Complex I activity and drives alpha-synuclein aggregation in models of Parkinson’s disease

Proteomimetic polymer blocks mitochondrial damage, rescues Huntington’s neurons, and slows onset of neuropathology in vivo

Associations of semaglutide with first‐time diagnosis of Alzheimer's disease in patients with type 2 diabetes: Target trial emulation using nationwide real‐world data in the US

Exploring temporal and sex-linked dysregulation in Alzheimer disease phosphoproteome

Author Correction: A selective inhibitor of mitofusin 1-βIIPKC association improves heart failure outcome in rats

Altered Metabolism in Motor Neuron Diseases: Mechanism and Potential Therapeutic Target

Temporal and Sex-Linked Protein Expression Dynamics in a Familial Model of Alzheimer’s Disease

ATAD3A oligomerization promotes neuropathology and cognitive deficits in Alzheimer’s disease models

A CHCHD6–APP axis connects amyloid and mitochondrial pathology in Alzheimer’s disease

The Putative Role of Astaxanthin in Neuroinflammation Modulation: Mechanisms and Therapeutic Potential

Altered Mitochondrial Protein Homeostasis and Proteinopathies

Gut–microbiota–microglia–brain interactions in Alzheimer’s disease: knowledge-based, multi-dimensional characterization

Mitochondrial Quality Control Strategies: Potential Therapeutic Targets for Neurodegenerative Diseases?

Adaptive translational pausing is a hallmark of the cellular response to severe environmental stress

Potential long‐term effect of tumor necrosis factor inhibitors on dementia risk: A propensity score matched retrospective cohort study in US veterans

Collaborators (7)

Rajesh Ramachandran

Marzieh Ayati

Mark Chance

Bindu Paul

George Perry

Maureen Dubreuil

Sunil Jamuna Tripathi

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