Cecilia Johansson

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Professor of Mucosal Immunology

Imperial College London

United Kingdom

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About

Professor Cecilia Johansson is a Professor of Mucosal Immunology at Imperial College London, UK. Her research primarily focuses on the immune responses to viral infections, with recent articles exploring the roles of natural killer cells, lung inflammation post-SARS-CoV-2 infection, and the immunogenicity of RNA vaccines. She investigates the mechanisms of innate immunity and the pathogenesis of respiratory viruses, contributing significantly to the understanding of host-pathogen interactions.

Recent Grants

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Lung resident memory CD8+ T cells during viral infections - generation and functionality regulated by localisation and type I IFNs

Open Date: 2021-05-31

Close Date: 2024-05-30

Grant: Close

Induction and role of type I and III interferons during SARS CoV2 infection

Open Date: 2020-11-10

Close Date: 2022-05-08

Grant: Close

The of type I interferons in the generation and maintenance of lung resident T cells

Open Date: 2019-08-01

Close Date: 2020-08-01

Grant: Close

Cancer and viral infections in the lung - a delicate balance of microenvironments

Open Date: 2019-05-01

Close Date: 2022-04-01

Grant: Close

The innate immune response in the lung- an ex vivo model to study human lung resident cells

Open Date: 2018-12-01

Close Date: 2021-11-01

Articles (13)

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2025

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2025

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2025

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2023

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2022

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2022

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2022

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2022

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2021

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2021

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2021

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Imperial College London

UNITED KINGDOM

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Cecilia Johansson

Research Interests

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About

Recent Grants

Articles (13)

Natural Killer Cells Are Dispensable for Virus Control in Rag2−/− Mice During Primary RSV Infection

Sustained Lung Inflammation Post‐SARS‐CoV‐2 Infection in Mice Is Associated with Increased Pulmonary T Cells

RORing for oral tolerance

Formulation, inflammation, and RNA sensing impact the immunogenicity of self-amplifying RNA vaccines

Levels of Influenza A Virus Defective Viral Genomes Determine Pathogenesis in the BALB/c Mouse Model

Type I interferon receptor signalling deficiency results in dysregulated innate immune responses to SARS-CoV-2 in mice.

The Use of Precision-Cut Lung Slices for Studying Innate Immunity to Viral Infections.

Type I interferons and MAVS signaling are necessary for tissue resident memory CD8+ T cell responses to RSV infection

Lentiviral and AAV-mediated expression of palivizumab offer protection against Respiratory Syncytial Virus infection

Neutrophils in respiratory viral infections

Rapidly Deployable Mouse Models of SARS-CoV-2 Infection Add Flexibility to the COVID-19 Toolbox

Neutrophilic inflammation in the respiratory mucosa predisposes to RSV infection

MAVS Deficiency Is Associated With a Reduced T Cell Response Upon Secondary RSV Infection in Mice

Collaborators (5)

Daniel Goldhill

Stephen Hyde

Leah Cuthbertson

John Tregoning

Wendy S. Barclay

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