Chad Swanson

King’s College London

United Kingdom

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Articles (14)

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2024

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2023

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2020

Collaborators (13)

Rui Galao

King’s College London

UNITED KINGDOM

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Wilhelm Furnon

University of Glasgow

UNITED KINGDOM

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Reiner Schulz

Senior Lecturer

King's College London - Guy's Campus

UNITED KINGDOM

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John Quinn

Professor

University of Liverpool

UNITED KINGDOM

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Daniel Goldhill

Lecturer

Royal Veterinary College

UNITED KINGDOM

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Giuditta De Lorenzo

University of Glasgow

UNITED KINGDOM

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Christopher Carroll

Lecturer in Human Genetics

St George’s, University of London

UNITED KINGDOM

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Michael P. Weekes

University of Cambridge

UNITED KINGDOM

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Richard Stanton

Professor of Virology

Cardiff University

UNITED KINGDOM

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Harry David Wilson

King’s College London

UNITED KINGDOM

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Alfredo Iacoangeli

King’s College London

UNITED KINGDOM

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Sam Wilson

GSK Professor of Microbial Pathogenesis

University of Cambridge

UNITED KINGDOM

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Blair L. Strang

University College London

UNITED KINGDOM

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Chad Swanson

Research Interests

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Articles (14)

Syn-CpG-Spacer: A Panel web app for synonymous recoding of viral genomes with CpG dinucleotides

Strain-Dependent Restriction of Human Cytomegalovirus by Zinc Finger Antiviral Proteins

A Nuclear Export Signal in KHNYN Required for Its Antiviral Activity Evolved as ZAP Emerged in Tetrapods

Inhibition of human cytomegalovirus replication by interferon alpha can involve multiple anti-viral factors

An assessment of bioinformatics tools for the detection of human endogenous retroviral insertions in short-read genome sequencing data.

The P681H Mutation in the Spike Glycoprotein of the Alpha Variant of SARS-CoV-2 Escapes IFITM Restriction and Is Necessary for Type I Interferon Resistance

TRIM25 and ZAP target the Ebola virus ribonucleoprotein complex to mediate interferon-induced restriction

RetroSnake: A modular pipeline to detect human endogenous retroviruses in genome sequencing data

Minimal impact of ZAP on lentiviral vector production and transduction efficiency

S-farnesylation is essential for antiviral activity of the long ZAP isoform against RNA viruses with diverse replication strategies

HIV-1 Vpr Induces Widespread Transcriptomic Changes in CD4 + T Cells Early Postinfection

The Polybasic Cleavage Site in SARS-CoV-2 Spike Modulates Viral Sensitivity to Type I Interferon and IFITM2

CpG Dinucleotides Inhibit HIV-1 Replication through Zinc Finger Antiviral Protein (ZAP)-Dependent and -Independent Mechanisms

SARS-CoV-2 Is Restricted by Zinc Finger Antiviral Protein despite Preadaptation to the Low-CpG Environment in Humans

Collaborators (13)

Rui Galao

Wilhelm Furnon

Reiner Schulz

John Quinn

Daniel Goldhill

Giuditta De Lorenzo

Christopher Carroll

Michael P. Weekes

Richard Stanton

Harry David Wilson

Alfredo Iacoangeli

Sam Wilson

Blair L. Strang

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