John Quinn

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University of Liverpool

United Kingdom

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John Quinn is a Professor at the University of Liverpool, United Kingdom. His research primarily focuses on neurological disorders, specifically the genetic determinants of amyotrophic lateral sclerosis (ALS), gene expression regulation, and the application of machine learning in identifying molecular subtypes of ALS. Recent contributions to the field include studies on SINE-VNTR-Alu elements, expression quantitative trait loci, and innovative genotyping platforms for diverse populations.

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Transcriptional regulation mediated by polymorphic domains termed variable number tandem repeats.

Open Date: 2001-04-01

Close Date: 2003-11-30

Articles (18)

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Collaborators (11)

Heather Mortiboys

Professor of Cellular Neuroscience and Metabolism

University of Sheffield

UNITED KINGDOM

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Ashley Jones

King’s College London

UNITED KINGDOM

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Sonja Scholz

National Institute of Neurological Disorders and Stroke

UNITED STATES

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Claire Troakes

King’s College London

UNITED KINGDOM

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Laura Ferraiuolo

Associate Professor

University of Sheffield

UNITED KINGDOM

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Chad Swanson

King’s College London

UNITED KINGDOM

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Thomas Julian

The University of Manchester

UNITED KINGDOM

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Sara Bandrés Ciga

Senior Staff Scientist | Head Training & Outreach

Centre for Addiction and Mental Health

UNITED STATES

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Johnathan Cooper‐Knock

UNITED KINGDOM

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jerzy kulski

University of Western Australia

AUSTRALIA

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Alfredo Iacoangeli

King’s College London

UNITED KINGDOM

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John Quinn

Research Interests

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Recent Grants

Articles (18)

The landscape of non-reference SINE-VNTR-Alus in amyotrophic lateral sclerosis.

Rare and common genetic determinants of mitochondrial function determine severity but not risk of amyotrophic lateral sclerosis

Exploring SVA Insertion Polymorphisms in Shaping Differential Gene Expressions in the Central Nervous System

A SINE-VNTR-Alu at the LRIG2 locus is associated with proximal and distal gene expression in CRISPR and population models

NeuroBooster Array: A Genome-Wide Genotyping Platform to Study Neurological Disorders Across Diverse Populations.

Gender-different effect of Src family kinases antagonism on photophobia and trigeminal ganglion activity.

A Genome-Wide Screen for the Exonisation of Reference SINE-VNTR-Alus and Their Expression in CNS Tissues of Individuals with Amyotrophic Lateral Sclerosis

Unsupervised machine learning identifies distinct ALS molecular subtypes in post-mortem motor cortex and blood expression data

Regulation of expression quantitative trait loci by SVA retrotransposons within the major histocompatibility complex.

Transcriptomic profiling of cerebrospinal fluid identifies ALS pathway enrichment and RNA biomarkers in MND individuals.

NeuroBooster Array: A Genome-Wide Genotyping Platform to Study Neurological Disorders Across Diverse Populations.

RetroSnake: A modular pipeline to detect human endogenous retroviruses in genome sequencing data

Characterisation of retrotransposon insertion polymorphisms in whole genome sequencing data from individuals with amyotrophic lateral sclerosis

A polymorphic transcriptional regulatory domain in the amyotrophic lateral sclerosis risk gene CFAP410 correlates with differential isoform expression

Src Family Kinases Facilitate the Crosstalk between CGRP and Cytokines in Sensitizing Trigeminal Ganglion via Transmitting CGRP Receptor/PKA Pathway

Non-reference genome transposable elements (TEs) have a significant impact on the progression of the Parkinson’s disease

Locus specific reduction of L1 expression in the cortices of individuals with amyotrophic lateral sclerosis.

Longitudinal intronic RNA-Seq analysis of Parkinson's disease patients reveals disease-specific nascent transcription.

Collaborators (11)

Heather Mortiboys

Ashley Jones

Sonja Scholz

Claire Troakes

Laura Ferraiuolo

Chad Swanson

Thomas Julian

Sara Bandrés Ciga

Johnathan Cooper‐Knock

jerzy kulski

Alfredo Iacoangeli

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