Charles Howe

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Professor

Mayo Clinic

United States

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About

Charles Howe is a Professor at Mayo Clinic in the United States. His research focuses on autoimmune encephalitis, neuroinflammation, and seizure disorders, as reflected in his recent publications. He has contributed to significant studies involving cytokine signatures, biospecimen collection in epilepsy, and the role of immune cells in neurological conditions.

Recent Grants

Grant: Close

New tools to study leukocyte infiltration into the CNS

Open Date: 2012-07-01

Close Date: 2014-06-30

Grant: Close

Brain-infiltrating inflammatory monocyte responses to acute virus infection

Open Date: 2009-08-01

Close Date: 2019-02-28

Articles (13)

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2025

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2024

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2024

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2023

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2023

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2023

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2022

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2022

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2022

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2022

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2022

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2022

Collaborators (16)

Albert Aboseif

Assistant Professor

Mayo Clinic

UNITED STATES

Charles Howe

Research Interests

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About

Recent Grants

Articles (13)

Cytokine, Chemokine, and Neurofilament Light Chain Signatures in LGI1 Autoimmune Encephalitis

New onset refractory status epilepticus: Summary of the NORSE Institute roundtable during the 2023 American Epilepsy Society meeting, Orlando, Florida

Defining the Spatial Resolution of Analyte Recovery during Microperfusion-Based Sampling of Brain Parenchyma

Review and standard operating procedures for collection of biospecimens and analysis of biomarkers in new onset refractory status epilepticus.

Intrathecal Dexamethasone in Febrile Infection–Related Epilepsy Syndrome

Drug‐resistant seizures associated with hyperinflammatory monocytes in FIRES

Development of an integrated microperfusion-EEG electrode for unbiased multimodal sampling of brain interstitial fluid and concurrent neural activity.

Rejuvenation of the aged brain immune cell landscape in mice through p16-positive senescent cell clearance

Preservation of antigen-specific responses in cryopreserved CD4<sup>+</sup> and CD8<sup>+</sup> T cells expanded with IL-2 and IL-7.

Peripheral blood mononuclear cell phenotype and function are maintained after overnight shipping of whole blood.

G9a Modulates Lipid Metabolism in CD4 T Cells to Regulate Intestinal Inflammation.

ISGylation is induced in neurons by demyelination driving ISG15-dependent microglial activation.

Septin-5 and -7-IgGs: Neurologic, Serologic, and Pathophysiologic Characteristics.

Collaborators (16)

Albert Aboseif

Aurelie Hanin

Krista Eschbach

Yi-Chen Lai

Diana Jurk

Gwen Lomberk

Yi Zhu

Xu Zhang

Gregory Day

Niyati Mehta

Margaret Gopaul

Michael Wilson

Alan Gonzalez-Suarez

Manuel B Braga Neto

Teneille Gofton

Adebowale Bamidele

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