Diana Jurk

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Associate Professor

Mayo Clinic Rochester

United States

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About

Diana Jurk is an Associate Professor at the Mayo Clinic Rochester in the United States. Her research primarily focuses on cellular senescence, aging, and age-related diseases, as evidenced by her recent publications. Key areas of her work include the effects of senescent cells on peripheral function and cognitive decline, telomere dysfunction in chronic liver disease, and the role of the blood-brain barrier in aging. Additionally, she actively contributes to guidelines and recommendations for studying cellular senescence in various tissues.

Recent Grants

Grant: Close

Investigating the mechanisms mediating lipid accumulation in the aging liver

Open Date: 2020-11-01

Close Date: 2022-10-01

Grant: Close

Does obesity exacerbate age-related cognitive decline via senescence?

Open Date: 2020-09-01

Close Date: 2025-05-01

Grant: Close

Targeting Cellular Senescence to Extend Healthspan

Open Date: 2020-08-01

Close Date: 2021-02-01

Grant: Close

Investigate the mechanisms mediating lipid accumulation in senescent cells in the liver

Open Date: 2020-06-01

Close Date: 2021-07-01

Grant: Close

Investigate the mechanisms mediating lipid accumulation in senescent cells in the liver

Open Date: 2020-06-01

Close Date: 2021-07-01

Articles (19)

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2024

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2024

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2024

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2023

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2023

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2022

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2022

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2022

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2022

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2022

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2022

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2022

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2021

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2021

Collaborators (19)

Laura J. Niedernhofer

UNITED STATES

Diana Jurk

Research Interests

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About

Recent Grants

Articles (19)

Author Correction: Apoptotic stress causes mtDNA release during senescence and drives the SASP (Nature, (2023), 622, 7983, (627-636), 10.1038/s41586-023-06621-4)

Guidelines for minimal information on cellular senescence experimentation in vivo

Senescent cell transplantation into the skin induces age-related peripheral dysfunction and cognitive decline.

Telomere dysfunction in chronic liver disease: The link from aging

Blood–brain barrier dysfunction in aging is mediated by brain endothelial senescence

SenNet recommendations for detecting senescent cells in different tissues.

Unlocking the Potential of Senolytic Compounds: Advancements, Opportunities, and Challenges in Ageing-Related Research.

Telomere dysfunction promotes cholangiocyte senescence and biliary fibrosis in Primary Sclerosing Cholangitis

Apoptotic stress causes mtDNA release during senescence and drives the SASP

A new face of old cells: An overview about the role of senescence and telomeres in inflammatory bowel diseases.

Rejuvenation of the aged brain immune cell landscape in mice through p16-positive senescent cell clearance

Orally-active, clinically-translatable senolytics restore α-Klotho in mice and humans

Telomere dysfunction in ageing and age-related diseases

Senolytic drugs: Beyond the promise and the hype.

A new gene set identifies senescent cells and predicts senescence-associated pathways across tissues

Characterization of cellular senescence in aging skeletal muscle

Short senolytic or senostatic interventions rescue progression of radiation-induced frailty and premature ageing in mice

Neutrophils induce paracrine telomere dysfunction and senescence in ROS‐dependent manner

Telmisartan prevents high-fat diet-induced neurovascular impairments and reduces anxiety-like behavior.

Collaborators (19)

Laura J. Niedernhofer

Peter Adams

Bernadette Carroll

Matthew M. Robinson

lino ferreira

Donato A. Rivas

Yi Zhu

Manuel Collado

Xu Zhang

Evripidis Gavathiotis

Dominik Saul

Paul Robson

Robert Huebert

Joao Passos

Elizabeth Schmidt

Nathan LeBrasseur

Alan Gonzalez-Suarez

Derek Mann

Charles Howe

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