Davi Mázala

Assistant Professor

Towson University

United States

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About

Davi Mázala is an Assistant Professor at Towson University in the United States. His research focuses on muscle health and disease, with recent publications addressing issues such as inflammation in Duchenne muscular dystrophy and the roles of specific muscle progenitor cells. Mázala's work also explores the mechanical stability of muscle fibers and the cellular responses to injury, contributing to a better understanding of muscle pathology and potential therapies.

Articles (11)

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2025

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2025

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2023

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2023

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2022

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2021

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2021

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2021

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2021

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2021

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2020

Collaborators (4)

James Novak

Assistant Professor

The George Washington University School of Medicine and Health SCiences

UNITED STATES

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Jyoti Jaiswal

Professor

The George Washington University School of Medicine and Health SCiences

UNITED STATES

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Alyson Fiorillo

Assistant Professor

Children's National Health System

UNITED STATES

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Heather Gordish-Dressman

George Washington University

UNITED STATES

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Davi Mázala

Research Interests

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Articles (11)

Failure to resolve inflammation contributes to juvenile onset cardiac damage in a mouse model of Duchenne muscular dystrophy

Acute Changes in Muscle Thickness and Quality Following Dynamic Bodyweight Exercise in Recreationally Trained Adults

Altered muscle niche contributes to myogenic deficit in the D2-mdx model of severe DMD

Single-cell transcriptomic analysis of the identity and function of fibro/adipogenic progenitors in healthy and dystrophic muscle

Absence of the Z-disc protein α-actinin-3 impairs the mechanical stability of Actn3KO mouse fast-twitch muscle fibres without altering their contractile properties or twitch kinetics

Human muscle stem cells are refractory to aging

Pathogenic role and therapeutic potential of fibro-adipogenic progenitors in muscle disease.

Endoplasmic reticulum maintains ion homeostasis required for plasma membrane repair

Coping with the calcium overload caused by cell injury: ER to the rescue.

Interrogation of Dystrophin and Dystroglycan Complex Protein Turnover After Exon Skipping Therapy

TGF-β–driven muscle degeneration and failed regeneration underlie disease onset in a DMD mouse model

Collaborators (4)

James Novak

Jyoti Jaiswal

Alyson Fiorillo

Heather Gordish-Dressman

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