David James

Professor

University of Sydney

Australia

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About

David James is a Professor at the University of Sydney, Australia. His research focuses on insulin signaling networks, metabolic reprogramming, and the genetic underpinnings of muscle insulin resistance. Recent publications include studies on glucose metabolism in brown adipose tissue and the structural aspects of Ras regulation. He employs systems genetics approaches to explore complex traits relevant to human disease.

Articles (19)

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2025

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2021

Collaborators (22)

Terrance Johns

Centre & Head of Oncogenic Signalling Laboratory

Telethon Kids Institute

AUSTRALIA

David James

Research Interests

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About

Articles (19)

The insulin signalling network

Analysis of Multiple Insulin Actions in Single Muscle Fibers From Insulin-Resistant Mice Reveals Selective Defect in Endogenous GLUT4 Translocation

Cold exposure stimulates cross-tissue metabolic rewiring to fuel glucose-dependent thermogenesis in brown adipose tissue

Super-stable lipid vacuoles endow cartilage with its shape and biomechanics

Unlocking metabolic insights with mouse genetic diversity

Dual Tracer Test to Measure Tissue-Specific Insulin Action in Individual Mice Identifies In Vivo Insulin Resistance Without Fasting Hyperinsulinemia

The genetic and dietary landscape of the muscle insulin signalling network

The genetic and dietary landscape of the muscle insulin signalling network

Systems genetics approaches for understanding complex traits with relevance for human disease

Structural insights into Ras regulation by SIN1

Dissecting the biology of mTORC1 beyond rapamycin

Bilirubin deficiency renders mice susceptible to hepatic steatosis in the absence of insulin resistance

The aetiology and molecular landscape of insulin resistance

Dynamic modelling of the PI3K/MTOR signalling network uncovers biphasic dependence of mTORC1 activity on the mTORC2 subunit SIN1

A hierarchical approach to removal of unwanted variation for large-scale metabolomics data

Global phosphoproteomics reveals DYRK1A regulates CDK1 activity in glioblastoma cells

BET inhibition blocks inflammation-induced cardiac dysfunction and SARS-CoV-2 infection.

Plasma Bile Acids More Closely Align With Insulin Resistance, Visceral and Hepatic Adiposity Than Total Adiposity.

Proteomics analysis of adipose depots after intermittent fasting reveals visceral fat preservation mechanisms.

Collaborators (22)

Terrance Johns

Richard Prince

Simon Foster

Mark Larance

Ariadna Recasens

Kristen Cooke

Sungyoung Shin

Michelle Digman

Chella Krishna Vadivel

Maneeshi Prasad

Stewart Masson

Ziyang Zhang

Lan Nguyen

Daniel J. Fazakerley

James Cantley

Cacang Suarna

Amy Merrill-Brugger

aldons lusis

JI WON OH

Eric O. Potma

Harry Cutler

Deanne Francis

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