EDUARDO MARTINEZ-NAVES

Professor

Universidad Complutense de Madrid

Spain

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About

Eduardo Martinez-Naves is a Professor at Universidad Complutense in Spain. His research interests include immunosuppressive therapies, chronic liver disease, and advancements in CAR-T cell therapy, as highlighted in his recent publications. He has contributed significantly to understanding the impact of various treatments on immune responses and liver health. Additionally, his work encompasses novel therapeutic approaches and the role of microbiota in health and disease.

Articles (14)

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2025

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2024

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2024

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2024

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2023

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2023

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2023

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2023

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2023

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2022

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2021

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2021

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2021

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2020

Collaborators (15)

María J Iraburu

Professor

Universidad de Navarra

SPAIN

EDUARDO MARTINEZ-NAVES

Research Interests

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About

Articles (14)

Immunosuppressive Therapy Modifies Anti-Spike IgG Subclasses Distribution After Four Doses of mRNA Vaccination in a Cohort of Kidney Transplant Recipients

State of the Art in CAR-T Cell Therapy for Solid Tumors: Is There a Sweeter Future?

Loss of <i>Cdkn1a</i> protects against MASLD alone or with alcohol intake by preserving lipid homeostasis.

Fecal microbiota transplantation from female donors restores gut permeability and reduces liver injury and inflammation in middle-aged male mice exposed to alcohol.

Novel therapeutic avenues for the study of chronic liver disease and regeneration: The foundation of the Iberoamerican Consortium for the study of liver Cirrhosis

Role of mTOR inhibitor in the cellular and humoral immune response to a booster dose of SARS-CoV-2 mRNA-1273 vaccine in kidney transplant recipients

Neuroblastoma RAS viral oncogene homolog (N-RAS) deficiency aggravates liver injury and fibrosis.

Expression of HMGCS2 in intestinal epithelial cells is downregulated in inflammatory bowel disease associated with endoplasmic reticulum stress.

Manufacture of Mesoporous Silicon Microparticles (MSMPs) as Adjuvants for Vaccine Delivery.

Genetic and pharmacological inhibition of XBP1 protects against APAP hepatotoxicity through the activation of autophagy.

Activation of the Unfolded Protein Response (UPR) Is Associated with Cholangiocellular Injury, Fibrosis and Carcinogenesis in an Experimental Model of Fibropolycystic Liver Disease

Fat: Quality, or Quantity? What Matters Most for the Progression of Metabolic Associated Fatty Liver Disease (MAFLD)

Abnormal Liver Function Test in Patients Infected with Coronavirus (SARS-CoV-2): A Retrospective Single-Center Study from Spain

Intestinal Epithelial Cell-Derived Extracellular Vesicles Modulate Hepatic Injury via the Gut-Liver Axis During Acute Alcohol Injury.

Collaborators (15)

María J Iraburu

Beatriz Salinas Rodríguez

Leonard Nelson

Roger Davis

Jose Regueiro

Ana Isabel Sanchez Fructuoso

Maite G. Fernández-Barrena

Carles Rentero

Rafael Bañares

Francisco Javier Cubero

Patricia Aspichueta

Yulia A. Nevzorova

August Vidal

Alejandro Hionides Gutierrez

Yolanda Campos-Martín

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