FERNANDO C. BALTANAS

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Assistant Professor

Universidad de Sevilla

Spain

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About

Fernando C. Baltanas is an Assistant Professor at the University of Seville, Spain. His research focuses on the role of SOS1 in cancer biology, particularly in KRAS-driven lung adenocarcinoma and chronic myelogenous leukemia. Recent publications include studies on SOS1 inhibitors and their therapeutic implications, as well as investigations into neurodegeneration linked to genetic mutations.

Recent Grants

Grant: Close

Terapia farmacológica combinada como nueva estrategia en el tratamiento del cáncer: opción terapéutica inicial o secundaria en mecanismos de resistencia adquirida

Open Date: 2023-01-01

Close Date: 2024-01-01

Grant: Close

Inhibición farmacológica de la ruta de señalización RAS/MAPK para el tratamiento del neuroblastoma recurrente y resistente a las terapias actuales

Open Date: 2022-01-01

Close Date: 2024-01-01

Grant: Close

Las proteínas Sos como dianas terapéuticas en el tratamiento del adenocarcinoma de pulmón causado por mutaciones en KRAS

Open Date: 2020-01-01

Close Date: 2021-01-01

Grant: Close

Las proteinas Sos como dianas terapeuticas en cancer

Open Date: 2018-01-01

Close Date: 2019-01-01

Articles (12)

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2025

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2025

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2023

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2023

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2022

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2021

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2021

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2021

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2021

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2021

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2021

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2020

Collaborators (2)

Anne Ridley

University of Bristol

UNITED KINGDOM

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Miguel Lafarga

Full Professor (Emeritus)

Universidad de Cantabria

SPAIN

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FERNANDO C. BALTANAS

Research Interests

Explore related searches

Contact this professor

About

Recent Grants

Articles (12)

SOS1 inhibitor BI-3406 shows in vivo antitumor activity akin to genetic ablation and synergizes with a KRAS G12D inhibitor in KRAS LUAD

Sos1 ablation alters focal adhesion dynamics and increases Mmp2/9-dependent gelatinase activity in primary mouse embryonic fibroblasts

Critical requirement of SOS1 for tumor development and microenvironment modulation in KRASG12D-driven lung adenocarcinoma

Advances in Molecular Research of Oncogenes

Critical Requirement of SOS1 for Development of BCR/ABL-Driven Chronic Myelogenous Leukemia

The Childhood-Onset Neurodegeneration with Cerebellar Atrophy (CONDCA) Disease Caused by AGTPBP1 Gene Mutations: The Purkinje Cell Degeneration Mouse as an Animal Model for the Study of this Human Disease

The Childhood-Onset Neurodegeneration with Cerebellar Atrophy (CONDCA) Disease Caused by AGTPBP1 Gene Mutations: The Purkinje Cell Degeneration Mouse as an Animal Model for the Study of this Human Disease

Critical requirement of SOS1 RAS-GEF function for mitochondrial dynamics, metabolism, and redox homeostasis

SOS2 Comes to the Fore: Differential Functionalities in Physiology and Pathology

Functional Specificity of the Members of the Sos Family of Ras-GEF Activators: Novel Role of Sos2 in Control of Epidermal Stem Cell Homeostasis

Functional Specificity of the Members of the Sos Family of Ras-GEF Activators: Novel Role of Sos2 in Control of Epidermal Stem Cell Homeostasis

SOS GEFs in health and disease

Collaborators (2)

Anne Ridley

Miguel Lafarga

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