Miguel Lafarga

Full Professor (Emeritus)

Universidad de Cantabria

Spain

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About

Professor Miguel Lafarga is a Full Professor (Emeritus) at the University of Cantabria-IDIVAL-CIBERNED in Spain. His research primarily focuses on neurobiology, including the response of neurons to sciatic nerve injury and the implications of oxidative stress in neurological conditions such as Down syndrome. He has contributed significantly to the study of epigenetic interactions and the development of innovative drug-screening platforms for treating brain metastasis.

Articles (11)

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2024

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2022

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2022

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2022

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2022

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2022

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2021

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2021

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2021

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2020

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2020

Collaborators (18)

Javier Munoz

Research Professor

Ikerbasque, Basque Foundation for Science

SPAIN

Miguel Lafarga

Research Interests

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Articles (11)

miR-30c-5p Gain and Loss of Function Modulate Sciatic Nerve Injury-Induced Nucleolar Stress Response in Dorsal Root Ganglia Neurons

Non-homogeneous dispersion of graphene in polyacrylonitrile substrates induces a migrastatic response and epithelial-like differentiation in MCF7 breast cancer cells

Oxidative-Stress-Associated Proteostasis Disturbances and Increased DNA Damage in the Hippocampal Granule Cells of the Ts65Dn Model of Down Syndrome

Identification of Epigenetic Interactions between MicroRNA-30c-5p and DNA Methyltransferases in Neuropathic Pain

Coaxial Synthesis of PEI-Based Nanocarriers of Encapsulated RNA-Therapeutics to Specifically Target Muscle Cells

A clinically compatible drug-screening platform based on organotypic cultures identifies vulnerabilities to prevent and treat brain metastasis

The Childhood-Onset Neurodegeneration with Cerebellar Atrophy (CONDCA) Disease Caused by AGTPBP1 Gene Mutations: The Purkinje Cell Degeneration Mouse as an Animal Model for the Study of this Human Disease

HSP70 chaperones RNA-free TDP-43 into anisotropic intranuclear liquid spherical shells

Nuclear reorganization in hippocampal granule cell neurons from a mouse model of down syndrome: Changes in chromatin configuration, nucleoli and cajal bodies

ALS-derived fibroblasts exhibit reduced proliferation rate, cytoplasmic TDP-43 aggregation and a higher susceptibility to DNA damage

Histiocytic hyperplasia with hemophagocytosis and acute alveolar damage in COVID-19 infection

Collaborators (18)

Javier Munoz

Maria T. Berciano

Haiyang Yu

Francisco Gil-Bea

Sandra Rodriguez-Perales

Maria Amor Hurle

Nazely Diban

Mónica Tramullas Fernández

Ane Urtiaga

Jose Rodriguez-Rey

SONIA MARTINEZ GONZALEZ

Carmen Martínez-Cué

FERNANDO C. BALTANAS

Osvaldo Graña Castro

Jose Fernandez Alen

Olga Tapia Martínez

Maria J Rivero

Damir Vareslija

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