Jae-Seok Roe

Associate Professor

Yonsei University

South Korea

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Jae-Seok Roe is an Associate Professor at Yonsei University in South Korea. His research areas include histone chaperones in relation to DNA replication and transcription, the targeting of histone demethylases for cancer therapy, and the roles of specific proteins in cancer metastasis. Recent publications highlight his work on enhancing the anti-tumor microenvironment and mechanisms of cancer cell proliferation and suppression.

Articles (13)

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2025

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2025

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2023

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2023

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2023

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2022

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2022

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2021

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2021

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2021

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2021

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2021

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2021

Collaborators (4)

Youngsoo Lee

Associate Professor

Ajou University School of Medicine

SOUTH KOREA

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Jong-ho Cha

Associate Professor

Inha University

SOUTH KOREA

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Matthew Vander Heiden

Massachusetts Institute of Technology

UNITED STATES

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TaeSoo Kim

Professor

Ewha Womans University

SOUTH KOREA

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Jae-Seok Roe

Research Interests

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Articles (13)

Histone chaperones coupled to DNA replication and transcription control divergent chromatin elements to maintain cell fate

Current advances and future directions in targeting histone demethylases for cancer therapy

<italic>Engrailed-1</italic> Promotes Pancreatic Cancer Metastasis

Reprogramming anchorage dependency by adherent-to-suspension transition promotes metastatic dissemination

Streamlined DNA-encoded small molecule library screening and validation for the discovery of novel chemotypes targeting BET proteins

LCK-Mediated RIPK3 Activation Controls Double-Positive Thymocyte Proliferation and Restrains Thymic Lymphoma by Regulating the PP2A-ERK Axis.

LCK-Mediated RIPK3 Activation Controls Double-Positive Thymocyte Proliferation and Restrains Thymic Lymphoma by Regulating the PP2A-ERK Axis

RIPK3 activation induces TRIM28 derepression in cancer cells and enhances the anti-tumor microenvironment.

RIPK3 activation induces TRIM28 derepression in cancer cells and enhances the anti-tumor microenvironment

GALNT3 suppresses lung cancer by inhibiting myeloid-derived suppressor cell infiltration and angiogenesis in a TNFR and c-MET pathway-dependent manner.

GALNT3 suppresses lung cancer by inhibiting myeloid-derived suppressor cell infiltration and angiogenesis in a TNFR and c-MET pathway-dependent manner

Core promoter activity contributes to chromatin-based regulation of internal cryptic promoters.

Core promoter activity contributes to chromatin-based regulation of internal cryptic promoters

Collaborators (4)

Youngsoo Lee

Jong-ho Cha

Matthew Vander Heiden

TaeSoo Kim

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