Jan-Willem Veening

Professor

University of Lausanne

Switzerland

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Jan-Willem Veening is a Professor at the University of Lausanne, Switzerland. His research focuses on bacterial genetics and antibiotic resistance, with recent publications exploring innovative methodologies like CRISPR interference in Streptococcus pneumoniae and Staphylococcus aureus. His work also investigates the role of immune responses in controlling bacterial infections and the organization of bacterial chromosomes.

Jan-Willem Veening

Research Interests

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Articles (25)

Genome-wide antibiotic-CRISPRi profiling identifies LiaR activation as a strategy to resensitize fluoroquinolone-resistant Streptococcus pneumoniae

Make-or-break prime editing for genome engineering in Streptococcus pneumoniae

HU promotes higher order chromosome organization and influences DNA replication rates in Streptococcus pneumoniae

PneumoBrowse 2: an integrated visual platform for curated genome annotation and multiomics data analysis of Streptococcus pneumoniae

Revitalizing antibiotic discovery and development through in vitro modelling of in-patient conditions.

Genome-wide CRISPRi screens for high-throughput fitness quantification and identification of determinants for dalbavancin susceptibility in Staphylococcus aureus

Triggering Toll-Like Receptor 5 Signaling During Pneumococcal Superinfection Prevents the Selection of Antibiotic Resistance

The function of CozE proteins is linked to lipoteichoic acid biosynthesis in Staphylococcus aureus.

An efficient in vivo -inducible CRISPR interference system for group A Streptococcus genetic analysis and pathogenesis studies

CRISPRi–TnSeq maps genome-wide interactions between essential and non-essential genes in bacteria

A conserved antigen induces respiratory Th17-mediated broad serotype protection against pneumococcal superinfection

Klebsiella pneumoniae OmpR facilitates lung infection through transcriptional regulation of key virulence factors

BactEXTRACT: an R Shiny app to quickly extract, plot and analyse bacterial growth and gene expression data

Uncovering the arsenal of class II bacteriocins in salivarius streptococci

Gaps in the wall: understanding cell wall biology to tackle amoxicillin resistance in Streptococcus pneumoniae

The <scp>ClpX</scp> chaperone and a hypermorphic <scp>FtsA</scp> variant with impaired self‐interaction are mutually compensatory for coordinating <i>Staphylococcus aureus</i> cell division

Pneumolysin promotes host cell necroptosis and bacterial competence during pneumococcal meningitis as shown by whole-animal dual RNA-seq

Mechanistic basis of choline import involved in teichoic acids and lipopolysaccharide modification

A Genome-Wide CRISPR Interference Screen Reveals an StkP-Mediated Connection between Cell Wall Integrity and Competence in Streptococcus salivarius

The acquisition of clinically relevant amoxicillin resistance in Streptococcus pneumoniae requires ordered horizontal gene transfer of four loci

Amoxicillin-resistant Streptococcus pneumoniae can be resensitized by targeting the mevalonate pathway as indicated by sCRilecs-seq

CRISPRi-seq for genome-wide fitness quantification in bacteria

CcrZ is a pneumococcal spatiotemporal cell cycle regulator that interacts with FtsZ and controls DNA replication by modulating the activity of DnaA

Switching off: the phenotypic transition to the uninduced state of the lactose uptake pathway

Harnessing CRISPR-Cas9 for Genome Editing in Streptococcus pneumoniae D39V

Collaborators (13)

Urs Jenal

Johannes Berg

Marek Basler

Christoph Dehio

Morten Kjos

Erida Gjini

Knut Drescher

Camilo Perez

Wilbert Bitter

Pascal Hols

Alexander Harms

Dorte Frees

Andreas Hierlemann

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