Jasmin Zaunschirm-Strutz

Head of degree programme

FH JOANNEUM University of Applied Sciences

Austria

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Jasmin Zaunschirm-Strutz is the Head of the Degree Programme at FH JOANNEUM University of Applied Sciences in Austria. Her recent research focuses on the role of microRNAs in metabolic disorders and their impact on neonatal health, specifically in the context of gestational diabetes and feto-placental angiogenesis. She has published work on topics including hyperglycemia-induced endothelial dysfunction and the effects of maternal metabolism on neonatal traits.

Articles (8)

Year:

2025

Year:

2023

Year:

2021

Year:

2021

Year:

2021

Year:

2020

Year:

2020

Year:

2020

Collaborators (7)

Ursula Hiden

Assoc. Prof.

Medical University of Graz

AUSTRIA

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Evelyn Jantscher-Krenn

Medical University of Graz

AUSTRIA

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Nicolle Müller

Jena University Hospital

GERMANY

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Akos Heinemann

Professor

Medical University of Graz

AUSTRIA

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Ruth Birner-Gruenberger

TU Wien

AUSTRIA

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Dilvin Semo

University Hospital Münster

GERMANY

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Christian Wadsack

Medical University of Graz

AUSTRIA

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Jasmin Zaunschirm-Strutz

Research Interests

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Articles (8)

MicroRNA profiling in umbilical cord plasma: links to maternal metabolism and neonatal metabolic and inflammatory traits

The Sodium-Glucose Co-Transporter 2 (SGLT2) Inhibitor Empagliflozin Reverses Hyperglycemia-Induced Monocyte and Endothelial Dysfunction Primarily through Glucose Transport-Independent but Redox-Dependent Mechanisms

SUCNR1 Is Expressed in Human Placenta and Mediates Angiogenesis: Significance in Gestational Diabetes

Transient Hyperglycemia and Hypoxia Induce Memory Effects in AngiomiR Expression Profiles of Feto-Placental Endothelial Cells.

Human Milk Oligosaccharides in Cord Blood Are Altered in Gestational Diabetes and Stimulate Feto-Placental Angiogenesis In Vitro.

Hyperglycemia-induced endothelial dysfunction is alleviated by thioredoxin mimetic peptides through the restoration of VEGFR-2-induced responses and improved cell survival

Maternal Overweight Downregulates MME (Neprilysin) in Feto-Placental Endothelial Cells and in Cord Blood

Sexual dimorphism of miRNA signatures in feto-placental endothelial cells is associated with altered barrier function and actin organization

Collaborators (7)

Ursula Hiden

Evelyn Jantscher-Krenn

Nicolle Müller

Akos Heinemann

Ruth Birner-Gruenberger

Dilvin Semo

Christian Wadsack

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