Ursula Hiden

Assoc. Prof.

Medical University of Graz

Austria

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Ursula Hiden is an Associate Professor at the Medical University of Graz, Austria. Her research focuses on maternal health, particularly in high-risk pregnancies, exploring topics like vascular adaptation, endothelial function, and the impact of maternal metabolism on neonatal outcomes. Recent publications include studies on the relationships between maternal factors and cardiovascular health, as well as investigations into the mechanisms of endothelial dysfunction associated with gestational diabetes.

Articles (16)

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2025

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2021

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2021

Collaborators (11)

Corina T. Madreiter-Sokolowski

Associate Professor

Medical University of Graz

AUSTRIA

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Reinier A. Boon

NETHERLANDS

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Evelyn Jantscher-Krenn

Medical University of Graz

AUSTRIA

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Nicolle Müller

Jena University Hospital

GERMANY

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Berthold Huppertz

Medical University of Graz

AUSTRIA

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Dilvin Semo

University Hospital Münster

GERMANY

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Karoline Mayer-Pickel

Medical University of Graz

AUSTRIA

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Jelena Krstic

Assistant Professor

Medical University of Graz

AUSTRIA

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Nandu Goswami

Medical University of Graz

AUSTRIA

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Jasmin Zaunschirm-Strutz

Head of degree programme

FH JOANNEUM University of Applied Sciences

AUSTRIA

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Martin Gauster

University Professor

Medical University of Graz

AUSTRIA

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Ursula Hiden

Research Interests

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Articles (16)

Maternal Vascular Adaptation in High-Risk Pregnancies: Effects of Early Smoking Cessation on Hemodynamic and Endothelial Function

Novel link between plasma bilirubin and anti-inflammatory miRNA profiles in follicular fluid of IVF patients

MicroRNA profiling in umbilical cord plasma: links to maternal metabolism and neonatal metabolic and inflammatory traits

Mitochondrial Dysfunction in Endothelial Progenitor Cells: Unraveling Insights from Vascular Endothelial Cells

Association of Circulating Neprilysin with BMI, Cardiovascular Health, and Kidney Function in High-Risk Pregnancies: A Pilot Study

The RhoA guanine exchange factor ABR: a glucose‐sensitive mediator of actin reorganization in feto‐placental arterial endothelial cells altered by gestational diabetes mellitus

Higher gestational weight gain delays wound healing and reduces expression of long non‐coding RNA KLRK1‐AS1 in neonatal endothelial progenitor cells

The Sodium-Glucose Co-Transporter 2 (SGLT2) Inhibitor Empagliflozin Reverses Hyperglycemia-Induced Monocyte and Endothelial Dysfunction Primarily through Glucose Transport-Independent but Redox-Dependent Mechanisms

(Dis)similarities between the Decidual and Tumor Microenvironment

Fetal sex and maternal fasting glucose affect neonatal cord blood-derived endothelial progenitor cells.

Transient Hyperglycemia and Hypoxia Induce Memory Effects in AngiomiR Expression Profiles of Feto-Placental Endothelial Cells.

Human Milk Oligosaccharides in Cord Blood Are Altered in Gestational Diabetes and Stimulate Feto-Placental Angiogenesis In Vitro.

Placental Endocrine Activity: Adaptation and Disruption of Maternal Glucose Metabolism in Pregnancy and the Influence of Fetal Sex.

Transcriptomic Remodelling of Fetal Endothelial Cells During Establishment of Inflammatory Memory.

Flow-through isolation of human first trimester umbilical cord endothelial cells

Type 1 Diabetes Mellitus and the First Trimester Placenta: Hyperglycemia-Induced Effects on Trophoblast Proliferation, Cell Cycle Regulators, and Invasion.

Collaborators (11)

Corina T. Madreiter-Sokolowski

Reinier A. Boon

Evelyn Jantscher-Krenn

Nicolle Müller

Berthold Huppertz

Dilvin Semo

Karoline Mayer-Pickel

Jelena Krstic

Nandu Goswami

Jasmin Zaunschirm-Strutz

Martin Gauster

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