John Griffin

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Adjunct Professor

University of California, San Diego

United States

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Recent Grants

Grant: Close

Regulation of Protein C Pathways

Open Date: 2018-06-01

Close Date: 2022-03-01

Grant: Close

The Protein C Pathway in Mitigation of Radiation-Induced Endothelial and Vascular Dysfunction

Open Date: 2017-06-01

Close Date: 2022-05-01

Grant: Close

Human exomics genotyping-driven discovery and characterization of proteins related to clinical thrombosis, blood coagulation and thrombin generation

Open Date: 2016-09-01

Close Date: 2020-08-01

Articles (10)

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2023

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2023

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2022

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2022

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2022

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2021

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2021

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2021

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2020

Year:

2020

Collaborators (10)

Heather Himburg

Associate Professor

Medical College of Wisconsin

UNITED STATES

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Usha R. Pendurthi

The University of Texas Health Science Center at Tyler

UNITED STATES

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Yaoxiang Li

Georgetown University

UNITED STATES

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University of North Carolina at Chapel Hill

UNITED STATES

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Hiroshi Deguchi

Scripps Research Institute

UNITED STATES

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John Griffin

Research Interests

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Recent Grants

Articles (10)

Analysis of the urinary metabolic profiles in irradiated rats treated with Activated Protein C (APC), a potential mitigator of radiation toxicity

Thrombin-mediated activation of PAR1 enhances doxorubicin-induced cardiac injury in mice

Full‐length plasma skeletal muscle myosin isoform deficiency is associated with coagulopathy in acutely injured patients

Activated Protein C Strengthens Cardiac Tolerance to Ischemic Insults in Aging

Protection of ischemic white matter and oligodendrocytes in mice by 3K3A-activated protein C

Factor VIIa induces extracellular vesicles from the endothelium: a potential mechanism for its hemostatic effect

PAR1 regulation of CXCL1 expression and neutrophil recruitment to the lung in mice infected with influenza A virus

Activated protein C and PAR1‐derived and PAR3‐derived peptides are anti‐inflammatory by suppressing macrophage NLRP3 inflammasomes

Procoagulant activities of skeletal muscle and cardiac myosins require both myosin protein and myosin-associated anionic phospholipids.

Skeletal muscle myosin and cardiac myosin attenuate heparin's antithrombin-dependent anticoagulant activity.

Collaborators (10)

Heather Himburg

Usha R. Pendurthi

Yaoxiang Li

Julia R. Coleman

Steven Grover

Marjan Boerma

Laurent O. Mosnier

DI Ren

Silvio Antoniak

Hiroshi Deguchi

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