John Max Michel

Auburn University

United States

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Articles (6)

Year:

2025

Year:

2025

Year:

2025

Year:

2024

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2024

Year:

2023

Collaborators (7)

Adam Sharples

Professor (Full) of Molecular Physiology and Epigenetics

Norwegian School of Sport Sciences

NORWAY

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Matt Stock

Assistant Professor

University of Central Florida

UNITED STATES

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David Hughes

Assistant Professor

Oklahoma Medical Research Foundation

UNITED STATES

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Vincent Dalbo

Central Queensland University

AUSTRALIA

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C. Brooks Mobley

Assistant Clinical Professor

UNITED STATES

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Michael D. Roberts

UNITED STATES

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Michael Kamal

Guest Lecturer

McMaster University

CANADA

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John Max Michel

Research Interests

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Articles (6)

Disuse and subsequent recovery resistance training affect skeletal muscle angiogenesis-related markers regardless of prior resistance training experience.

Skeletal muscle atrophy induced by aging and disuse atrophy are strongly associated with the upregulation of the endoplasmic stress protein CHOP in rats.

Is there a volume saturation point for muscle hypertrophy in resistance-trained individuals? A narrative review

Exercise is medicine, or exercise with medicine? Comparative effects on Na+,K+‐ATPase regulation

Skeletal muscle myosin heavy chain fragmentation as a potential marker of protein degradation in response to resistance training and disuse atrophy

Different Resistance Exercise Loading Paradigms Similarly Affect Skeletal Muscle Gene Expression Patterns of Myostatin-Related Targets and mTORC1 Signaling Markers

Collaborators (7)

Adam Sharples

Matt Stock

David Hughes

Vincent Dalbo

C. Brooks Mobley

Michael D. Roberts

Michael Kamal

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