Joseph Chambers

Assistant Research Professor

University of Cambridge

United Kingdom

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Joseph Chambers is an Assistant Research Professor at the University of Cambridge, UK. His research focuses on various aspects of cellular and molecular biology, particularly in relation to endoplasmic reticulum dynamics, protein trafficking, and the mechanisms underlying disease pathologies such as alpha-1 antitrypsin deficiency. He has published several articles exploring topics including drug delivery efficacy in cancer therapy and the regulation of neuronal regeneration.

Articles (10)

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2025

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2024

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2024

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2024

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2023

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2023

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2022

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2022

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2022

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2022

Collaborators (7)

Mosab Ali Awadelkareem

University of Oxford

UNITED KINGDOM

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Stefan J. Marciniak

University of Cambridge

UNITED KINGDOM

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Ioanna Mela

University of Cambridge

UNITED KINGDOM

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David Ron

Lab Head (Professor)

University of Cambridge

UNITED KINGDOM

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REBECCA VOORHEES

Assistant Professor

California Institute of Technology

UNITED STATES

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Qing Jun Meng

The University of Manchester

UNITED KINGDOM

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Ismael Lopez-Duarte

Assistant Professor

Universidad Complutense de Madrid

SPAIN

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Joseph Chambers

Research Interests

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Articles (10)

Selection for somatic escape variants in SERPINA1 in the liver of patients with alpha-1 antitrypsin deficiency

A quantitative pipeline to assess secretion of human leptin coding variants reveals mechanisms underlying leptin deficiencies.

Endoplasmic reticulum morphology regulation by RTN4 modulates neuronal regeneration by curbing luminal transport.

Enhancing Drug Delivery Efficacy Through Bilayer Coating of Zirconium-Based Metal–Organic Frameworks: Sustained Release and Improved Chemical Stability and Cellular Uptake for Cancer Therapy

Macromolecular condensation buffers intracellular water potential.

Synthetic Par polarity induces cytoskeleton asymmetry in unpolarized mammalian cells.

Z-α 1 -antitrypsin polymers impose molecular filtration in the endoplasmic reticulum after undergoing phase transition to a solid state

Pharmacological targeting of endoplasmic reticulum stress in disease

Novel insights into surfactant protein C trafficking revealed through the study of a pathogenic mutant

Full-length antithrombin frameshift variant with aberrant C-terminus causes endoplasmic reticulum retention with a dominant-negative effect

Collaborators (7)

Mosab Ali Awadelkareem

Stefan J. Marciniak

Ioanna Mela

David Ron

REBECCA VOORHEES

Qing Jun Meng

Ismael Lopez-Duarte

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