Ki Ho Park

Assistant Professor

University of Virginia

United States

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About

Dr. Ki Ho Park is an Assistant Professor at the University of Virginia, United States. His research primarily focuses on cardiovascular injury, regenerative medicine, and the role of biomolecules in tissue healing, as evidenced by his recent articles on monoclonal antibodies, traumatic brain injury, and stem cell technology. His work aims to develop innovative therapies for improving heart and kidney function, as well as addressing complications related to vascular interventions.

Articles (19)

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2025

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2020

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2020

Collaborators (13)

Paul Janssen

Professor

Ohio State University

UNITED STATES

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Pei-Hui Lin

Research Associate Professor

The Ohio State University Wexner Medical Center

UNITED STATES

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Ang Li

Assistant Professor of Research

The University of Texas at Arlington

UNITED STATES

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Elizabeth Kirby

Assistant Professor

Ohio State University

UNITED STATES

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Xuejun Wang

Professor and MD/PhD Program Director

South Dakota State University

UNITED STATES

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Onur Kanisicak

Associate Professor

Ohio State University

UNITED STATES

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Takashi Murayama

Juntendo Daigaku

JAPAN

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Hannahong1 Ong

Ohio State University

UNITED STATES

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Willa Hsueh

Professor

Ohio State University Medical Center

UNITED STATES

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Lei Cao

Ohio State University

UNITED STATES

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Haichang Li

Assistant Professor

Ohio State University

UNITED STATES

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Yigang Wang

University of Cincinnati

UNITED STATES

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Albert DAHDAH

Karolinska Institutet

SWEDEN

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Ki Ho Park

Research Interests

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About

Articles (19)

Humanized Monoclonal Antibody Against Citrullinated Histone H3 Attenuates Myocardial Injury and Prevents Heart Failure in Rodent Models

Treatment with MG53 ameliorates traumatic brain injury–associated acute kidney injury

Development of an Ophthalmic Hydrogel to Deliver MG53 and Promote Corneal Wound Healing

Nano-based perivascular intervention sustains a nine-month long-term suppression of intimal hyperplasia in vein grafts

Bioorthogonal non-canonical amino acid tagging to track transplanted human induced pluripotent stem cell-specific proteome

MG53’s non-physiologic interaction with insulin receptor: lack of effect on insulin-stimulated Akt phosphorylation in muscle, heart and liver tissues

The role of 6-phosphogluconate dehydrogenase in vascular smooth muscle cell phenotypic switching and angioplasty-induced intimal hyperplasia

Activation of MG53 Enhances Cell Survival and Engraftment of Human Induced Pluripotent Stem Cell-Derived Cardiomyocytes in Injured Hearts

Quantification of autophagy flux in isolated mouse skeletal muscle fibers with overexpression of fluorescent protein mCherry-EGFP-LC3

UCHL1 protects against ischemic heart injury via activating HIF-1α signal pathway

MG53 preserves mitochondrial integrity of cardiomyocytes during ischemia reperfusion-induced oxidative stress

MG53 Inhibits Necroptosis Through Ubiquitination-Dependent RIPK1 Degradation for Cardiac Protection Following Ischemia/Reperfusion Injury

Retention of the NLRP3 Inflammasome–Primed Neutrophils in the Bone Marrow Is Essential for Myocardial Infarction–Induced Granulopoiesis

CRISPR activation of endogenous genes reprograms fibroblasts into cardiovascular progenitor cells for myocardial infarction therapy

MG53 Preserves Neuromuscular Junction Integrity and Alleviates ALS Disease Progression

MG53 suppresses NF-κB activation to mitigate age-related heart failure

MG53 suppresses interferon-β and inflammation via regulation of ryanodine receptor-mediated intracellular calcium signaling

MG53 Does Not Manifest the Development of Diabetes in db/db Mice

TRIC-A Channel Maintains Store Calcium Handling by Interacting With Type 2 Ryanodine Receptor in Cardiac Muscle

Collaborators (13)

Paul Janssen

Pei-Hui Lin

Ang Li

Elizabeth Kirby

Xuejun Wang

Onur Kanisicak

Takashi Murayama

Hannahong1 Ong

Willa Hsueh

Lei Cao

Haichang Li

Yigang Wang

Albert DAHDAH

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