Kohji Mori

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Associate Professor

Osaka University

Japan

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About

Dr. Kohji Mori is an Associate Professor at Osaka University, Japan. His research focuses on neurodegenerative diseases, including frontotemporal lobar degeneration and Alzheimer's disease, with recent studies addressing therapeutic targets and the use of machine learning in diagnostic processes. He is actively involved in consortium efforts for familial frontotemporal dementia in Japan and has published extensively on the molecular mechanisms underlying these conditions.

Recent Grants

Grant: Close

Elucidation of molecular mechanisms of repeat-associated non-AUG translation and its role in neurodegenerative diseases

Open Date: 2020-11-19

Close Date: 2025-03-31

Grant: Close

前頭側頭型認知症における「RNA代謝リレー障害」仮説の実証

Open Date: 2020-04-01

Close Date: 2023-03-31

Grant: Close

Paradigm convergence of non-AUG translation and cellular stress response

Open Date: 2018-06-29

Close Date: 2021-03-31

Grant: Close

Identification of a metabolic pathway for aberrant RNA in frontotemporal dementia and its disruption

Open Date: 2017-04-01

Close Date: 2020-03-31

Grant: Close

異常RNAの分解経路に着目した前頭側頭型認知症の病態解析

Open Date: 2016-08-26

Close Date: 2018-03-31

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2022

Collaborators (6)

Yoshitaka Nagai

JAPAN

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Toshihiko Sugiki

Kitasato University

JAPAN

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Nicholas Kanaan

Associate Professor

Michigan State University College of Human Medicine

UNITED STATES

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Yuto Satake

Assistant Professor

Osaka University

JAPAN

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MASAHIRO HATA

Osaka University

JAPAN

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Hideki Kanemoto

Associate Professor

Osaka University

JAPAN

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Kohji Mori

Research Interests

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About

Recent Grants

Articles (24)

iPSC screening identifies CACNA2D2 as a potential therapeutic target for FTLD-Tau

Screening of Aβ and phosphorylated tau status in the cerebrospinal fluid through machine learning analysis of portable electroencephalography data

Psychiatric‐onset neuronal intranuclear inclusion disease in a psychiatry‐based dementia‐enriched cohort in Japan

Nation-wide Japanese FTD consortium FTLD-J: Utility of case review meetings

Motor involvement in frontotemporal lobar degeneration with TAR DNA-binding protein of 43 kDa type C

PABPC1 mediates degradation of C9orf72-FTLD/ALS GGGGCC repeat RNA

eIF5 stimulates the CUG initiation of RAN translation of poly-GA dipeptide repeat protein (DPR) in C9orf72 FTLD/ALS

Gene-gene functional relationships in Alzheimer ' s disease: CELF1 regulates KLC1 alternative splicing

Utilizing portable electroencephalography to screen for pathology of Alzheimer's disease: a methodological advancement in diagnosis of neurodegenerative diseases

Protocols for identifying endogenous interactors of RNA-binding proteins in mammalian cells using the peroxidase APEX2 biotin-labeling method

Correction to: Bidirectional transcripts of the expanded C9orf72 hexanucleotide repeat are translated into aggregating dipeptide repeat proteins (Acta Neuropathologica, (2013), 126, 6, (881-893), 10.1007/s00401-013-1189-3)

Gene-gene functional relationships in Alzheimer's disease: CELF1 regulates KLC1 alternative splicing

Cerebrospinal fluid amyloid beta with amyloid positron emission tomography concordance rates in a heterogeneous group of patients including late‐onset psychotic disorders: a retrospective cross‐sectional study

A heterozygous splicing variant IVS9-7A > T in intron 9 of the MAPT gene in a patient with right-temporal variant frontotemporal dementia with atypical 4 repeat tauopathy

FUS regulates RAN translation through modulating the G-quadruplex structure of GGGGCC repeat RNA in C9orf72-linked ALS/FTD

FUS regulates RAN translation through modulating the G-quadruplex structure of GGGGCC repeat RNA in C9orf72-linked ALS/FTD

Aspects of degradation and translation of the expanded C9orf72 hexanucleotide repeat RNA.

Therapeutic reduction of GGGGCC repeat RNA levels by hnRNPA3 suppresses neurodegeneration in Drosophila models of C9orf72-linked ALS/FTD

Ribavirin Treatment for Severe Schizophrenia with Anti-Borna Disease Virus 1 Antibodies 30 Years after Onset

Characteristics of very late-onset schizophrenia-like psychosis classified with the biomarkers for Alzheimer's disease: a retrospective cross-sectional study.

FUS regulates RAN translation through modulating the G-quadruplex structure of GGGGCC repeat RNA in <i>C9orf72</i>-linked ALS/FTD

FUS regulates RAN translation through modulating the G-quadruplex structure of GGGGCC repeat RNA in C9orf72-linked ALS/FTD

RNA Dysmetabolism and Repeat-Associated Non-AUG Translation in Frontotemporal Lobar Degeneration/Amyotrophic Lateral Sclerosis due to <i>C9orf72</i> Hexanucleotide Repeat Expansion.

Biological basis and psychiatric symptoms in frontotemporal dementia

Collaborators (6)

Yoshitaka Nagai

Toshihiko Sugiki

Nicholas Kanaan

Yuto Satake

MASAHIRO HATA

Hideki Kanemoto

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