Laetitia Marcadet

Professionnelle de recherche

Laval University

Canada

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About

Laetitia Marcadet is a Research Professional at Université Laval in Canada. Her research primarily focuses on neurodegenerative diseases, particularly amyotrophic lateral sclerosis, and includes implications in cardiology and immunology. Recent articles highlight her work on lipid droplet targeting in ALS and the roles of RANK/RANKL/OPG in muscular health and disease.

Articles (7)

Year:

2025

Year:

2024

Year:

2023

Year:

2023

Year:

2022

Year:

2021

Year:

2020

Collaborators (5)

Sandra Minotti

McGill University

CANADA

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Silvia Pozzi

Assistant Professor

Laval University

CANADA

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Jérôme Frenette

Laval University

CANADA

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A. Russell Tupling

Professor

University of Waterloo

CANADA

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Caroline Vance

King’s College London

UNITED KINGDOM

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Laetitia Marcadet

Research Interests

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Articles (7)

Targeting lipid droplets in FUS-linked amyotrophic lateral sclerosis mitigates neuronal and astrocytic lipotoxicity

Reversal of cognitive deficits in FUS<sup>R521G</sup> amyotrophic lateral sclerosis mice by arimoclomol and a class I histone deacetylase inhibitor independent of heat shock protein induction.

RANKL Inhibition Reduces Cardiac Hypertrophy in mdx Mice and Possibly in Children with Duchenne Muscular Dystrophy

Neuronal dysfunction caused by FUSR521G promotes ALS-associated phenotypes that are attenuated by NF-κB inhibition

The Roles of RANK/RANKL/OPG in Cardiac, Skeletal, and Smooth Muscles in Health and Disease

TLR7 Signaling Drives the Development of Sjögren’s Syndrome

Muscle weakness and selective muscle atrophy in osteoprotegerin-deficient mice

Collaborators (5)

Sandra Minotti

Silvia Pozzi

Jérôme Frenette

A. Russell Tupling

Caroline Vance

Make your next move.

Laetitia Marcadet

Research Interests

Explore related searches

Contact this professor

About

Articles (7)

Targeting lipid droplets in FUS-linked amyotrophic lateral sclerosis mitigates neuronal and astrocytic lipotoxicity

Reversal of cognitive deficits in FUS&lt;sup&gt;R521G&lt;/sup&gt; amyotrophic lateral sclerosis mice by arimoclomol and a class I histone deacetylase inhibitor independent of heat shock protein induction.

RANKL Inhibition Reduces Cardiac Hypertrophy in mdx Mice and Possibly in Children with Duchenne Muscular Dystrophy

Neuronal dysfunction caused by FUSR521G promotes ALS-associated phenotypes that are attenuated by NF-κB inhibition

The Roles of RANK/RANKL/OPG in Cardiac, Skeletal, and Smooth Muscles in Health and Disease

TLR7 Signaling Drives the Development of Sjögren’s Syndrome

Muscle weakness and selective muscle atrophy in osteoprotegerin-deficient mice

Collaborators (5)

Sandra Minotti

Silvia Pozzi

Jérôme Frenette

A. Russell Tupling

Caroline Vance

Make your next move.

Reversal of cognitive deficits in FUS<sup>R521G</sup> amyotrophic lateral sclerosis mice by arimoclomol and a class I histone deacetylase inhibitor independent of heat shock protein induction.