Lauren Vaughn

Research Assistant Professor

University of South Carolina

United States

Research Interests

Explore related searches

Professors in United States

Contact this professor

Get a free first email built on one of their papers, ready for you to edit and send yourself.

About

Lauren Vaughn is a Research Assistant Professor at the University of South Carolina, United States. Her research focuses on neuronal injury and regeneration, with recent articles exploring topics such as axon regeneration, synaptic transmission, and the role of dsRNA-binding proteins in signaling pathways. Vaughn's work emphasizes the molecular mechanisms underlying neuronal growth and the stress response in the context of neurological disorders.

Articles (8)

Year:

2025

Year:

2024

Year:

2022

Year:

2022

Year:

2022

Year:

2021

Year:

2020

Year:

2020

Collaborators (4)

Jeffery L Twiss

University of South Carolina

UNITED STATES

View details

Jennifer Dulin

Associate Professor

Texas A&M University

UNITED STATES

View details

Pabitra Sahoo

Research Assistant Professor

University of South Carolina

UNITED STATES

View details

Patricia Ward

Assistant Professor

Emory University School of Medicine

UNITED STATES

View details

ApplyKite TwinLearns as you apply

Make your next move.

Prepare your application

Review drafts before you send them.

Lauren Vaughn

Research Interests

Explore related searches

Contact this professor

About

Articles (8)

Disruption of G3BP1 granules promotes mammalian CNS and PNS axon regeneration

Neuronal Injury Model Divulges Differences in Dendrite and Axonal Function and Regeneration in Adults

Intra-axonal translation of Khsrp mRNA slows axon regeneration by destabilizing localized mRNAs

KHSRP loss increases neuronal growth and synaptic transmission and alters memory consolidation through RNA stabilization.

DYT-PRKRA Mutation P222L Enhances PACT’s Stimulatory Activity on Type I Interferon Induction

Opposite actions of two dsRNA-binding proteins PACT and TRBP on RIG-I mediated signaling.

Dystonia 16 (DYT16) mutations in PACT cause dysregulated PKR activation and eIF2α signaling leading to a compromised stress response.

Toll-like Receptor (TLR)-induced Rasgef1b expression in macrophages is regulated by NF-κB through its proximal promoter.

Collaborators (4)

Jeffery L Twiss

Jennifer Dulin

Pabitra Sahoo

Patricia Ward

Make your next move.