Lu Le

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Full Proffesor (with tenure)

University of Texas Southwestern Medical School

United States

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About

Lu Le is a Full Professor with tenure at the University of Texas Southwestern Medical School in the United States. His research focuses on the pathogenesis of neurofibromatosis type 1, particularly examining biological sex and gender identity, tumor growth mechanisms, and the role of epithelial stem cells. He has authored several articles addressing topics such as the impact of TGFβ signaling, skin tumor dynamics, and innovative imaging techniques in the assessment of the disease.

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Articles (20)

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2025

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2021

Collaborators (16)

Sang Lee

Johns Hopkins University School of Medicine

UNITED STATES

Lu Le

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About

Recent Grants

Articles (20)

Hair follicle epithelial stem cells contribute to interfollicular epidermis during homeostasis

Accounting for Biological Sex and Gender Identity in the Pathogenesis, Artistic Depictions, and Quality of Life in Neurofibromatosis Type 1

Using Durometers to Quantify Stiffness as an Outcome Measure for Cutaneous Neurofibroma in Neurofibromatosis Type 1

Post-Translational Regulation of Transcription Factor KROX20 in Epithelial Stem Cells

TGFβ dependent signaling drives tumor growth and aberrant extracellular matrix dynamics in NF1-associated plexiform neurofibroma

STING activation reprograms the microenvironment to sensitize NF1-related malignant peripheral nerve sheath tumors for immunotherapy

Transcription factor KROX20 marks epithelial stem cell ancestors for hair follicle formation

Malignant melanoma developing in a pre-existing cutaneous neurofibroma from a patient with neurofibromatosis type 1

Multiparametric whole-body MRI of patients with neurofibromatosis type I: spectrum of imaging findings

Effect of NFX-179 MEK inhibitor on cutaneous neurofibromas in persons with neurofibromatosis type 1

A Call for Discovery and Therapeutic Development for Cutaneous Neurofibromas

Cutaneous Neurofibroma Heterogeneity: Factors that Influence Tumor Burden in Neurofibromatosis Type 1

Existing and Developing Preclinical Models for Neurofibromatosis Type 1−Related Cutaneous Neurofibromas

Basement membrane proteins in extracellular matrix characterize NF1 neurofibroma development and response to MEK inhibitor

Whole Central and Peripheral Nervous System Mice Dissection

Neuronal hyperexcitability drives central and peripheral nervous system tumor progression in models of neurofibromatosis-1

Malignant peripheral nerve sheath tumor: models, biology, and translation

Scoliosis in Neurofibromatosis Type 1 on Whole-Body Magnetic Resonance Imaging: Frequency and Association With Intraspinal and Paraspinal Tumors

Neurofibromin and suppression of tumorigenesis: beyond the GAP

The Research Techniques Made Simple Series: Lasting and Future Impact on Investigative Dermatology

Collaborators (16)

Sang Lee

Kavita Y. Sarin

Ross Cagan

Renée McKay

Carlos G Romo

Pierre Wolkenstein

Kristen Kelly

Chao Xing

David Gutmann

Jaishri Blakeley

Zhuan Zhou

Steven Angus

Michelle Monje

Kevin Grimes

Muzlifah Haniffa

Sara Brown

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