Luca Fava

Associate Professor

Università degli Studi di Torino

Italy

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About

Dr. Luca Fava is an Associate Professor at the Università degli Studi di Trento, Italy. His research focuses on cancer biology, inflammation, and cell signaling pathways, with recent publications addressing topics such as the role of caspase-2 in cell death, the impact of cancer-related mutations, and innovative CRISPR-Cas technologies. He is actively engaged in advancing the understanding of molecular mechanisms underlying various cancers and potential therapeutic strategies.

Articles (13)

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2024

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2024

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2024

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2024

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2024

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2023

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2023

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2023

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2022

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2022

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2021

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2021

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2020

Collaborators (8)

Graziano Lolli

Associate professor

University of Trento

ITALY

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Angel Barco

Professor of Research - Lab head

Instituto de Neurociencias

SPAIN

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Alessandra BISIO

Assistant Professor

University of Trento

ITALY

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Bart Westendorp

Utrecht University

NETHERLANDS

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Alessio Locallo

Københavns Universitet

DENMARK

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Andreas Villunger

Medical University of Innsbruck

AUSTRIA

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Maurizio Viscardi

Istituto Zooprofilattico Sperimentale dell'Umbria e delle Marche

ITALY

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Sabrina Ghetti

Max Planck Institute of Molecular Physiology

GERMANY

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Luca Fava

Research Interests

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Articles (13)

Caspase-2 kills cells with extra centrosomes.

Novel, soluble 3-heteroaryl-substituted tanshinone mimics attenuate the inflammatory response in murine macrophages.

Cyclin C promotes development and progression of B-cell acute lymphoblastic leukemia by counteracting p53-mediated stress responses.

Targeting ATP2B1 impairs PI3K/Akt/FOXO signaling and reduces SARS-COV-2 infection and replication.

Loss-of-Function but Not Gain-of-Function Properties of Mutant TP53 Are Critical for the Proliferation, Survival, and Metastasis of a Broad Range of Cancer Cells.

Extra centrosomes induce PIDD1‐mediated inflammation and immunosurveillance

PLK1 promotes the mitotic surveillance pathway by controlling cytosolic 53BP1 availability

Triap1 upregulation promotes escape from mitotic-slippage-induced G1 arrest.

Allele-specific genomic data elucidate the role of somatic gain and copy-number neutral loss of heterozygosity in cancer

Generation of corrected hiPSC clones from a Cornelia de Lange Syndrome (CdLS) patient through CRISPR-Cas-based technology.

CRISPR/Cas9 ribonucleoprotein-mediated knockin generation in hTERT-RPE1 cells

The PIDDosome: centrosome guardian and backup on the DNA damage response

E2F-Family Members Engage the PIDDosome to Limit Hepatocyte Ploidy in Liver Development and Regeneration.

Collaborators (8)

Graziano Lolli

Angel Barco

Alessandra BISIO

Bart Westendorp

Alessio Locallo

Andreas Villunger

Maurizio Viscardi

Sabrina Ghetti

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