Bart Westendorp

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Utrecht University

Netherlands

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Recent Grants

Grant: Open

Snatching DNA from neighbouring cells: a critical mechanism to promote cancer growth and therapy resistance?

Open Date: 2022-05-01

Close Date: 2033-05-01

Grant: Close

Unraveling cell-to-cell heterogeneity in the molecular defense against DNA replication stress with single cell technology.

Open Date: 2019-05-01

Close Date: 2023-05-01

Articles (12)

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2024

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2024

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2024

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2023

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2022

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2022

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2021

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2021

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2020

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2020

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2020

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2020

Collaborators (10)

Bart L. Haagmans

Principal Investigator / Associate Professor - PhD

Erasmus MC

NETHERLANDS

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Luca Fava

Associate Professor

Università degli Studi di Torino

ITALY

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Alain de Bruin

Professor in Pathobiology

Utrecht University

NETHERLANDS

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Shusil Pandit

University of Southern California Keck School of Medicine

UNITED STATES

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Daphne Stapels

Utrecht University

NETHERLANDS

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Albert Heck

Utrecht University

NETHERLANDS

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Karin Strijbis

Associate Professor

Utrecht University

NETHERLANDS

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Frank Riemers

Utrecht University

NETHERLANDS

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Jos van Putten

Utrecht University

NETHERLANDS

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Tim Veth

University of Washington

UNITED STATES

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Bart Westendorp

Research Interests

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Recent Grants

Articles (12)

Assessment of Kinome-Wide Activity Remodeling upon Picornavirus Infection

MUC13 negatively regulates tight junction proteins and intestinal epithelial barrier integrity via protein kinase C

Mesenchymal stromal/stem cells promote intestinal epithelium regeneration after chemotherapy-induced damage

Glycosylated extracellular mucin domains protect against SARS-CoV-2 infection at the respiratory surface

Oncogenic RAS sensitizes cells to drug-induced replication stress via transcriptional silencing of P53

Mechanisms used by cancer cells to tolerate drug-induced replication stress

Collection of cells for single-cell RNA sequencing using high-resolution fluorescence microscopy

Atypical E2Fs either Counteract or Cooperate with RB during Tumorigenesis Depending on Tissue Context

E2F-Family Members Engage the PIDDosome to Limit Hepatocyte Ploidy in Liver Development and Regeneration.

Excessive E2F Transcription in Single Cancer Cells Precludes Transient Cell-Cycle Exit after DNA Damage

PIDDosome‐induced p53‐dependent ploidy restriction facilitates hepatocarcinogenesis

E2F7 is a potent inhibitor of liver tumor growth in adult mice.

Collaborators (10)

Bart L. Haagmans

Luca Fava

Alain de Bruin

Shusil Pandit

Daphne Stapels

Albert Heck

Karin Strijbis

Frank Riemers

Jos van Putten

Tim Veth

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