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Marc Aurel Busche

Group Leader

University of Basel

Switzerland

Research Interests

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Professors in Switzerland

Articles (9)

Year:

2026

Year:

2025

Year:

2025

Year:

2025

Year:

2025

Year:

2024

Year:

2024

Year:

2024

Year:

2023

Collaborators (10)

Samuel Harris

Senior Research Fellow

University College London

UNITED KINGDOM

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David Long

Professor in Paediatric Nephrology and Wellcome Trust Investigator in Science

University College London Institute of Child Health

UNITED KINGDOM

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Raymond J. Dolan

Director

UCL Institute of Neurology, University College London

UNITED KINGDOM

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Arne Skerra

GERMANY

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David Dupret

University of Oxford

UNITED KINGDOM

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Demelza Smeeth

Senior Research Fellow

University College London

UNITED KINGDOM

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Hannah Mitchell

Senior Lecturer

Queen's University Belfast

UNITED KINGDOM

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Andreas Neef

GERMANY

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Menna Clatworthy

Professor of Translational Immunology

University of Cambridge

UNITED KINGDOM

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Christine Grienberger

Assistant Professor of Biology

Brandeis University

UNITED STATES

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Marc Aurel Busche

Research Interests

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Articles (9)

Aberrant tau accumulation caused by MAPT mutations induces early pathological changes in axonal transport that are rescued by p38α inhibition

Alzheimer’s disease patient-derived high-molecular-weight tau impairs bursting in hippocampal neurons

Organ-specific features of human kidney lymphatics are disrupted in chronic transplant rejection.

Selectively vulnerable deep cortical layer 5/6 fast-spiking interneurons in Alzheimer's disease models in vivo

The amyloid precursor family of proteins in excitatory neurons are essential for regulating cortico-hippocampal circuit dynamics<i> in</i><i> vivo</i>

β-amyloid monomer scavenging by an anticalin protein prevents neuronal hyperactivity in mouse models of Alzheimer’s Disease

PolyGR and polyPR knock-in mice reveal a conserved neuroprotective extracellular matrix signature in <i>C9orf72</i> ALS/FTD neurons

Selective suppression of oligodendrocyte-derived amyloid beta rescues neuronal dysfunction in Alzheimer's Disease

PolyGR and polyPR knock-in mice reveal a conserved neuroprotective extracellular matrix signature in<italic> C9orf72</italic> ALS/FTD neurons

Collaborators (10)

Samuel Harris

David Long

Raymond J. Dolan

Arne Skerra

David Dupret

Demelza Smeeth

Hannah Mitchell

Andreas Neef

Menna Clatworthy

Christine Grienberger

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