Marco A. Calzado

Associate Professor

Universidad de Córdoba

Spain

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Dr. Marco A. Calzado is an Associate Professor at the University of Córdoba, Spain. His research focuses on cancer biology, metabolic disorders, and the mechanisms of cellular signaling, as evidenced by his recent publications on topics such as the regulation of tumor growth and the impact of metabolic pathways on endocrine functions. He explores novel therapeutic targets and the evolutionary aspects of metabolic diseases in his work.

Articles (16)

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2025

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2024

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2024

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2024

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2024

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2023

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2023

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2022

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2021

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2020

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2020

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2020

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2020

Collaborators (11)

Mitchell Chen

Clinical Lecturer

Imperial College London

UNITED KINGDOM

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Francisco Manuel Ortuño

Assistant Professor

Universidad de Granada

SPAIN

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Viktoria Haug

University Hospital Frankfurt

GERMANY

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Sumeet Hindocha

Clinical Lecturer

University College London

UNITED KINGDOM

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Markus E. Diefenbacher

GERMANY

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David Garcia Galiano

Assistant Professor

Universidad de Córdoba

SPAIN

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Martin Eilers

Professor

University of Würzburg

GERMANY

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Laureano de la Vega

University of Dundee

UNITED KINGDOM

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Felix Christian Eduard Vogel

Deutsches Krebsforschungszentrum

GERMANY

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Rafael Pineda Reyes

Associate professor

Universidad de Córdoba

SPAIN

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Christian Münch

Goethe University Frankfurt

GERMANY

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Marco A. Calzado

Research Interests

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Articles (16)

A novel feedback loop between DYRK2 and USP28 regulates cancer homeostasis and DNA damage signaling

The evolutionary conserved miR-137/325 tandem mediates obesity-induced hypogonadism and metabolic comorbidities by repressing hypothalamic kisspeptin

Deep representation learning of tissue metabolome and computed tomography annotates NSCLC classification and prognosis

Suppression of metastatic organ colonization and antiangiogenic activity of the orally bioavailable lipid raft-targeted alkylphospholipid edelfosine

Localization, traffic and function of Rab34 in adipocyte lipid and endocrine functions

USP28 controls SREBP2 and the mevalonate pathway to drive tumour growth in squamous cancer.

FBXW7 tumor suppressor regulation by dual specificity tyrosine-regulated kinase 2.

Altered splicing machinery in lung carcinoids unveils NOVA1, PRPF8 and SRSF10 as novel candidates to understand tumor biology and expand biomarker discovery

USP28 enables oncogenic transformation of respiratory cells and its inhibition potentiates molecular therapy targeting mutant EGFR, BRAF and PI3K.

A versatile workflow to integrate RNA-seq genomic and transcriptomic data into mechanistic models of signaling pathways

A novel CDC25A/DYRK2 regulatory switch modulates cell cycle and survival

Betulinic Acid Hydroxamate is Neuroprotective and Induces Protein Phosphatase 2A-Dependent HIF-1α Stabilization and Post-transcriptional Dephosphorylation of Prolyl Hydrolase 2

Updating dual-specificity tyrosine-phosphorylation-regulated kinase 2 (DYRK2): molecular basis, functions and role in diseases

Betulinic acid hydroxamate prevents colonic inflammation and fibrosis in murine models of inflammatory bowel disease.

Phosphorylation-dependent regulation of the NOTCH1 intracellular domain by dual-specificity tyrosine-regulated kinase 2

Δ<sup>9</sup> -Tetrahydrocannabinolic acid alleviates collagen-induced arthritis: Role of PPARγ and CB<sub>1</sub> receptors.

Collaborators (11)

Mitchell Chen

Francisco Manuel Ortuño

Viktoria Haug

Sumeet Hindocha

Markus E. Diefenbacher

David Garcia Galiano

Martin Eilers

Laureano de la Vega

Felix Christian Eduard Vogel

Rafael Pineda Reyes

Christian Münch

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