Mary Wilson

Professor

Iowa State University

United States

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About

Mary Wilson is a Professor at the University of Iowa. Her research focuses on immunology and infectious diseases, with particular emphasis on Guillain-Barre Syndrome, visceral leishmaniasis, and the interactions of neutrophils with various pathogens. Recent publications explore cellular pathways and immune responses in infectious contexts, contributing significantly to the understanding of these conditions. She is located in the United States.

Articles (16)

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2025

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2025

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2025

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2024

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2024

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2024

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2023

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2022

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2022

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2021

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2021

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2021

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2021

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2021

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2021

Collaborators (3)

Jenna Guthmiller

Assistant Professor

University of Colorado Health

UNITED STATES

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Rahul Vijay

Assistant Professor

Rosalind Franklin University of Medicine and Science

UNITED STATES

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Noah Butler

Associate Professor

Iowa State University

UNITED STATES

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Mary Wilson

Research Interests

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Articles (16)

Altered Cellular Pathways in the Blood of Patients With Guillain-Barre Syndrome.

The circulating plasma microRNA signature in human visceral leishmaniasis

Neutrophil-<i>Leishmania infantum</i> Interaction Induces Neutrophil Extracellular Traps, DAMPs, and Inflammatory Molecule Release.

Contrasting Phenotypes of Neutrophils During Asymptomatic Versus Symptomatic Leishmania braziliensis Infection

Impaired signaling pathways on Berardinelli–Seip congenital lipodystrophy macrophages during Leishmania infantum infection

Apolipoprotein E Is Upregulated in Blood and Circulating Monocytes of Indian Patients With Visceral Leishmaniasis

Hierarchical spatiotemporal modeling of human visceral leishmaniasis in Rio Grande do Norte, Brazil

NLRP12-expressing dendritic cells mediate both dissemination of infection and adaptive immune responses in visceral leishmaniasis

Leishmania braziliensis causing human disease in Northeast Brazil presents loci with genotypes in long-term equilibrium

Leishmania braziliensis causing human disease in Northeast Brazil presents loci with genotypes in long-term equilibrium

Correction for Kiser et al., “The Inflammatory Effects of Dietary Lipids Regulate Growth of Parasites during Visceral Leishmaniasis”

Erratum: Correction for Kiser et al., "The Inflammatory Effects of Dietary Lipids Regulate Growth of Parasites during Visceral Leishmaniasis" (mSphere (2021) 6 4 (e0042321))

The Inflammatory Effects of Dietary Lipids Regulate Growth of Parasites during Visceral Leishmaniasis.

The Inflammatory Effects of Dietary Lipids Regulate Growth of Parasites during Visceral Leishmaniasis

Hemolysis-associated phosphatidylserine exposure promotes polyclonal plasmablast differentiation.

Anti-Interleukin-10 Unleashes Transcriptional Response to Leishmanial Antigens in Visceral Leishmaniasis Patients.

Collaborators (3)

Jenna Guthmiller

Rahul Vijay

Noah Butler

Make your next move.

Mary Wilson

Research Interests

Explore related searches

Contact this professor

About

Articles (16)

Altered Cellular Pathways in the Blood of Patients With Guillain-Barre Syndrome.

The circulating plasma microRNA signature in human visceral leishmaniasis

Neutrophil-&lt;i&gt;Leishmania infantum&lt;/i&gt; Interaction Induces Neutrophil Extracellular Traps, DAMPs, and Inflammatory Molecule Release.

Contrasting Phenotypes of Neutrophils During Asymptomatic Versus Symptomatic Leishmania braziliensis Infection

Impaired signaling pathways on Berardinelli–Seip congenital lipodystrophy macrophages during Leishmania infantum infection

Apolipoprotein E Is Upregulated in Blood and Circulating Monocytes of Indian Patients With Visceral Leishmaniasis

Hierarchical spatiotemporal modeling of human visceral leishmaniasis in Rio Grande do Norte, Brazil

NLRP12-expressing dendritic cells mediate both dissemination of infection and adaptive immune responses in visceral leishmaniasis

Leishmania braziliensis causing human disease in Northeast Brazil presents loci with genotypes in long-term equilibrium

Leishmania braziliensis causing human disease in Northeast Brazil presents loci with genotypes in long-term equilibrium

Correction for Kiser et al., “The Inflammatory Effects of Dietary Lipids Regulate Growth of Parasites during Visceral Leishmaniasis”

Erratum: Correction for Kiser et al., "The Inflammatory Effects of Dietary Lipids Regulate Growth of Parasites during Visceral Leishmaniasis" (mSphere (2021) 6 4 (e0042321))

The Inflammatory Effects of Dietary Lipids Regulate Growth of Parasites during Visceral Leishmaniasis.

The Inflammatory Effects of Dietary Lipids Regulate Growth of Parasites during Visceral Leishmaniasis

Hemolysis-associated phosphatidylserine exposure promotes polyclonal plasmablast differentiation.

Anti-Interleukin-10 Unleashes Transcriptional Response to Leishmanial Antigens in Visceral Leishmaniasis Patients.

Collaborators (3)

Jenna Guthmiller

Rahul Vijay

Noah Butler

Make your next move.

Neutrophil-<i>Leishmania infantum</i> Interaction Induces Neutrophil Extracellular Traps, DAMPs, and Inflammatory Molecule Release.