MASAHIRO SHUDA

Assistant Professor

University of Pittsburgh

United States

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Masahiro Shuda is an Assistant Professor at the University of Pittsburgh, USA. His research areas include viral oncology, with a particular focus on the mechanisms of virus-induced tumors, such as those related to Merkel cell polyomavirus and Epstein–Barr virus. Recent articles authored by him explore various aspects of tumorigenicity, cancer cell stemness, and novel therapeutic vulnerabilities in relation to gastrointestinal stromal tumors.

Articles (10)

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2025

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2024

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2023

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2023

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2023

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2022

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2022

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2022

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2022

Year:

2020

Collaborators (7)

Patrick Moore

UNITED STATES

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Zandrea Ambrose

Associate Professor

University of Pittsburgh

UNITED STATES

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Daniel Hoffmann

Professor

Universität Duisburg-Essen

GERMANY

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Rui Sun

University of Pittsburgh

UNITED STATES

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Yuan Chang

Distinguished Professor

University of Pittsburgh

UNITED STATES

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Donna Stolz

Associate Professor

University of Pittsburgh

UNITED STATES

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Shou-Jiang Gao

UNITED STATES

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MASAHIRO SHUDA

Research Interests

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Articles (10)

Disruption of ATR Signaling by Epstein–Barr Virus Latent Membrane Protein 1 Sensitizes Nasopharyngeal Carcinoma Cells to Cisplatin

Merkel cell polyomavirus pan‐T antigen knockdown reduces cancer cell stemness and promotes neural differentiation independent of RB1

Methyltransferase-independent function of enhancer of zeste homologue 2 maintains tumorigenicity induced by human oncogenic papillomavirus and polyomavirus

Merkel Cell Polyomavirus T Antigen–Mediated Reprogramming in Adult Merkel Cell Progenitors

Mitotic CDK1 and 4E-BP1 II: A single phosphomimetic mutation in 4E-BP1 induces glucose intolerance in mice.

Replication Kinetics for a Reporter Merkel Cell Polyomavirus

Merkel cell polyomavirus small T antigen is a viral transcription activator that is essential for viral genome maintenance

Targeting the translational machinery in gastrointestinal stromal tumors (GIST): a new therapeutic vulnerability.

Construction and characterization of two SARS-CoV-2 minigenome replicon systems.

MCPyV Large T Antigen-Induced Atonal Homolog 1 Is a Lineage-Dependency Oncogene in Merkel Cell Carcinoma

Collaborators (7)

Patrick Moore

Zandrea Ambrose

Daniel Hoffmann

Rui Sun

Yuan Chang

Donna Stolz

Shou-Jiang Gao

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