Masahito Ikawa

Distinguished Professor

Osaka University

Japan

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Masahito Ikawa is a Distinguished Professor at Osaka University, Japan. His research primarily focuses on male reproductive biology, with recent publications covering areas such as sperm motility, meiotic repair mechanisms, and novel approaches to fertility treatment. Notable work includes the exploration of genetic factors influencing male reproduction and the development of ex vivo models for studying embryogenesis and implantation.

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2021

Collaborators (16)

Daiji Kiyozumi

Visiting associate professor

JAPAN

Masahito Ikawa

Research Interests

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Articles (20)

TEX38 localizes ZDHHC19 to the plasma membrane and regulates sperm head morphogenesis in mice

CRISPR/Cas9‐mediated genome editing reveals seven testis‐enriched transmembrane glycoproteins dispensable for male fertility in mice

Mouse genome engineering uncovers 18 genes dispensable for male reproduction

Allosteric inhibition of the IZUMO1–JUNO fertilization complex by the naturally occurring antisperm antibody OBF13

Compensation for X-linked Pdha1 silencing by Pdha2 is essential for meiotic double-strand break repair in spermatogenesis

An ex vivo uterine system captures implantation, embryogenesis, and trophoblast invasion via maternal-embryonic signaling

The absence of both RIBC1 and RIBC2 induces decreased sperm motility and litter size in male mice

Therapeutic Potential of Emricasan, a Pan-Caspase Inhibitor, in Reducing Cell Death and Extracellular Matrix Accumulation in Fuchs Endothelial Corneal Dystrophy

CRISPR/Cas9-mediated genome editing reveals seven testis-enriched transmembrane glycoproteins dispensable for male fertility in mice

Inhibition of ROS1 activity with lorlatinib reversibly suppresses fertility in male mice.

Astrocytic cAMP modulates memory via synaptic plasticity

FAM71F1 binds to RAB2A and RAB2B and is essential for acrosome formation and male fertility in mice

FAM209 associates with DPY19L2 and is required for sperm acrosome biogenesis and fertility in mice.

SPATA33 localizes calcineurin to the mitochondria and regulates sperm motility in mice

KCTD19 and its associated protein ZFP541 are independently essential for meiosis in male mice

The conserved fertility factor SPACA4/Bouncer has divergent modes of action in vertebrate fertilization

Precise CAG repeat contraction in a Huntington’s Disease mouse model is enabled by gene editing with SpCas9-NG

Lens–specific conditional knockout of tropomyosin 1 gene in mice causes abnormal fiber differentiation and lens opacity

Protocol for isolation of spermatids from mouse testes

ARMC12 regulates spatiotemporal mitochondrial dynamics during spermiogenesis and is required for male fertility

Collaborators (16)

Daiji Kiyozumi

Taichi NODA

Takayuki Koyano

Haruhiko Miyata

Zhiwen Zhou

Dhirendra P Singh

Kazuki Okamoto

Makoto Matsuyama

Yonggang Lu

Chang Rok Kim

Keisuke Shimada

kentaro shimada

Yasushi Hirota

Shaogeng Tang

Ryo Sakasai

Yuhkoh Satouh

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