Michael Sigal

Professor for Translational Gastrointestinal Oncology

Charité - Universitätsmedizin Berlin

Germany

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About

Michael Sigal is a Professor for Translational Gastrointestinal Oncology at Charité Universitätsmedizin Berlin, Germany. His research primarily focuses on the implications of Helicobacter pylori in gastric carcinogenesis, biliary microbial patterns in primary sclerosing cholangitis, and the long-term gastrointestinal effects of COVID-19. Recent publications highlight his contributions to understanding the role of microbiota, prognostic biomarkers in neuroendocrine neoplasms, and the mechanisms of gastrointestinal tissue regeneration.

Articles (10)

Year:

2023

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2023

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2023

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2023

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2023

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2023

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2023

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2022

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2022

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2022

Collaborators (8)

Julian Heuberger

Charité - Universitätsmedizin Berlin

GERMANY

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Andreas Edel

ChariteCentrum 7 fur Anasthesiologie OP-Management und Intensivmedizin

GERMANY

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Lichao liu

Charité - Universitätsmedizin Berlin

GERMANY

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Raphael Mohr

Charité - Universitätsmedizin Berlin

GERMANY

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Kimberly Hartl

Charité - Universitätsmedizin Berlin

GERMANY

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Klaus Rajewsky

Senior Group Leader and Professor

Max Delbrück Centrum für Molekulare Medizin Berlin Buch

GERMANY

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Felix Balzer

Charité - Universitätsmedizin Berlin

GERMANY

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Kai Markus Schneider

Assistant Professor, Research group leader

RWTH Aachen University

GERMANY

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Michael Sigal

Research Interests

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Articles (10)

Association of Helicobacter Pylori Positivity with Risk of Disease and Mortality.

Biliary microbial patterns in primary sclerosing cholangitis are linked to poorer transplant-free survival.

Establishment of gastrointestinal assembloids to study the interplay between epithelial crypts and their mesenchymal niche.

Hepatobiliary long-term consequences of COVID-19: dramatically increased rate of secondary sclerosing cholangitis in critically ill COVID-19 patients.

Osteopontin is a prognostic circulating biomarker in patients with neuroendocrine neoplasms.

Short-term mucosal disruption enables colibactin-producing E. coli to cause long-term perturbation of colonic homeostasis.

Unwelcome guests - the role of gland-associated Helicobacter pylori infection in gastric carcinogenesis.

Epigenetic modifier balances Mapk and Wnt signalling in differentiation of goblet and Paneth cells

R-spondin/YAP axis promotes gastric oxyntic gland regeneration and Helicobacter pylori–associated metaplasia in mice

Gastric stem cells promote inflammation and gland remodeling in response to Helicobacter pylori via Rspo3‐Lgr4 axis

Collaborators (8)

Julian Heuberger

Andreas Edel

Lichao liu

Raphael Mohr

Kimberly Hartl

Klaus Rajewsky

Felix Balzer

Kai Markus Schneider

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