Minghui Wang

Associate Professor

Icahn School of Medicine at Mount Sinai

United States

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About

Minghui Wang is an Associate Professor at the Icahn School of Medicine at Mount Sinai in the United States. His research encompasses areas such as neurodegenerative diseases, including Alzheimer's and Parkinson's, as well as the cellular mechanisms underlying breast cancer resilience. Recent publications highlight his work on cell signaling in intervertebral disc repair, molecular profiling of the substantia nigra, and the regulatory networks associated with insulin resistance.

Articles (14)

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2025

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2022

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2022

Collaborators (11)

Roland Friedel

Associate Professor

Icahn School of Medicine at Mount Sinai

UNITED STATES

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Chrystian Junqueira Alves

Assistant Professor

Icahn School of Medicine at Mount Sinai

UNITED STATES

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Philip Hasel

NYU Langone Medical Center

UNITED STATES

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Allen Pan

Mount Sinai School of Medicine

UNITED STATES

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Assistant professor

Wake Forest School of Medicine

UNITED STATES

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Minghui Wang

Research Interests

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About

Articles (14)

TNFR1-mediated senescence and lack of TNFR2-signaling limit human intervertebral disc cell repair potential in degenerative conditions

Regulation of cell distancing in peri-plaque glial nets by Plexin-B1 affects glial activation and amyloid compaction in Alzheimer's disease.

Molecular profiling of human substantia nigra identifies diverse neuron types associated with vulnerability in Parkinson’s disease

Multi-omic atlas of the parahippocampal gyrus in Alzheimer’s disease

Autophagy enables microglia to engage amyloid plaques and prevents microglial senescence

Multi-omic integration reveals cell-type-specific regulatory networks of insulin resistance in distinct ancestry populations.

Convergent coexpression of autism-associated genes suggests some novel risk genes may not be detectable in large-scale genetic studies

Genome-wide methylomic regulation of multiscale gene networks in Alzheimer's disease

Molecular differences in brain regional vulnerability to aging between males and females

Multiomics Analyses Identify Proline Endopeptidase-Like Protein As a Key Regulator of Protein Trafficking, a Pathway Underlying Alzheimer's Disease Pathogenesis

Tuberous sclerosis complex-1 (TSC1) contributes to selective neuronal vulnerability in Alzheimer's disease

Dysfunction of ubiquitin protein ligase MYCBP2 leads to cell resilience in human breast cancers

Dual-Specificity Protein Phosphatase 4 (DUSP4) Overexpression Improves Learning Behavior Selectively in Female 5xFAD Mice, and Reduces β-Amyloid Load in Males and Females.

Microglial INPP5D limits plaque formation and glial reactivity in the PSAPP mouse model of Alzheimer's disease.

Collaborators (11)

Roland Friedel

Chrystian Junqueira Alves

Philip Hasel

Allen Pan

Xiaodong Zhu

INSUP CHOI

Jean-Vianney Haure-Mirande

Stephen Salton

ERMING WANG

Kurt Farrell

Swapan Das

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