Mohamed Bentires-Alj

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Professor of experimental surgical oncology

University of Basel

Switzerland

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Professor Mohamed Bentires-Alj is a faculty member at the University of Basel, Switzerland. His research areas encompass experimental surgical oncology, with a focus on breast cancer metastasis, cellular environments, and the impact of microbiota on viral infections. He has contributed to various studies on the mechanisms of tumor biology, including the role of epigenetic factors and immunophenotyping in cancer progressions.

Recent Grants

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Deconstructing the evolution of metastasis “EVOMET”

Open Date: 2021-01-01

Close Date: 2024-01-01

Grant: Close

Molecular mechanisms underlying normal and neoplastic mammary stem cells, progression to metastasis and resistance to therapy

Open Date: 2019-12-01

Close Date: 2023-11-30

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Effects of glucocorticoids in metastatic breast cancer

Open Date: 2019-07-01

Close Date: 2021-06-01

Grant: Close

Personalized Oncology 2019 conference

Open Date: 2019-05-01

Close Date: 2019-07-31

Grant: Close

Assessing the effects of protein tyrosine phosphatase SHP2 on immune cells in models of metastatic breast cancer

Open Date: 2019-03-01

Close Date: 2022-02-01

Mohamed Bentires-Alj

Research Interests

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Recent Grants

Articles (21)

Microenvironmental Regulation of Dormancy in Breast Cancer Metastasis: "An Ally that Changes Allegiances".

Cigarette smoke-induced disordered microbiota aggravates the severity of influenza A virus infection

15th Annual ENBDC Meeting: How do Cellular Potency, Microenvironment and Natural Rhythms Influence Mammary Gland Biology and Breast Cancer?

Optimized full-spectrum flow cytometry panel for deep immunophenotyping of murine lungs.

BRCA promoter methylation in triple-negative breast cancer is preserved in xenograft models and represents a potential therapeutic marker for PARP inhibitors.

Imagine beyond: recent breakthroughs and next challenges in mammary gland biology and breast cancer research

Nicotinamide N‐methyltransferase sustains a core epigenetic program that promotes metastatic colonization in breast cancer

Deletion of SNX9 alleviates CD8 T cell exhaustion for effective cellular cancer immunotherapy.

Single-cell Analysis Reveals Inter- and Intratumour Heterogeneity in Metastatic Breast Cancer.

PI3K inhibition circumvents resistance to SHP2 blockade in metastatic triple-negative breast cancer.

ERK3/MAPK6 dictates CDC42/RAC1 activity and ARP2/3-dependent actin polymerization.

N-acetylcysteine overcomes NF1 loss-driven resistance to PI3Kα inhibition in breast cancer.

Live slow-frozen human tumor tissues viable for 2D, 3D, ex vivo cultures and single-cell RNAseq

Thirteenth Annual ENBDC Workshop: Methods in Mammary Gland Biology and Breast Cancer

Feed‐forward loops between metastatic cancer cells and their microenvironment—the stage of escalation

Protein Tyrosine Phosphatase SHP2 Controls Interleukin-8 Expression in Breast Cancer Cells

Phenotypic plasticity during metastatic colonization.

A high-throughput drug screen reveals means to differentiate triple-negative breast cancer.

Hepatic stellate cells suppress NK cell-sustained breast cancer dormancy.

The NFIB-ERO1A axis promotes breast cancer metastatic colonization of disseminated tumour cells.

Reply to: Hippo signalling maintains ER expression and ER+ breast cancer growth

Collaborators (17)

Beata Katarzyna Kaczyńska

Andrea Banfi

Michael Krauthammer

Alfred Zippelius

Loïc Sauteur

Alexander Schmidt

Robert Clarke

Kirsten Mertz

Romina Marone

Lukas Jeker

Christian Beisel

Gaetana Restivo

Marco P Alves

Salvatore Piscuoglio

Joao C Guimaraes

Marja Mikkola

Marcus Vetter

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