Najib El-Sayed

Professor

University of Maryland

United States

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Professor Najib El-Sayed is a faculty member at the University of Maryland, USA. His research areas encompass cutaneous leishmaniasis, virulence regulators in Streptococcus pyogenes, and urinary tract infections associated with Pseudomonas aeruginosa. His recent publications explore topics such as the molecular signatures of healing, pathogen interactions, and genomic analyses related to host colonization and infection resistance.

Articles (14)

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2025

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2024

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2024

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2024

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2022

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2022

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2022

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2021

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2021

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2021

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2020

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2020

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Professor

Brunel University London

UNITED KINGDOM

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Najib El-Sayed

Research Interests

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About

Articles (14)

Innate biosignature of treatment failure in human cutaneous leishmaniasis

Characterization of the Entner-Doudoroff pathway in Pseudomonas aeruginosa catheter-associated urinary tract infections

The Streptococcus pyogenes stand-alone regulator RofA exhibits characteristics of a PRD-containing virulence regulator

Consolidation of a Molecular Signature of Healing in Cutaneous Leishmaniasis Is Achieved during the First 10 Days of Treatment

Catheter-associated urinary tract infection by Pseudomonas aeruginosa progresses through acute and chronic phases of infection

Loss of rpoE Encoding the δ-Factor of RNA Polymerase Impacts Pathophysiology of the Streptococcus pyogenes M1T1 Strain 5448

Genomic Analyses Identify Manganese Homeostasis as a Driver of Group B Streptococcal Vaginal Colonization

PIWI silencing mechanism involving the retrotransposon nimbus orchestrates resistance to infection with Schistosoma mansoni in the snail vector, Biomphalaria glabrata.

Early Leukocyte Responses in <i>Ex-Vivo</i> Models of Healing and Non-Healing Human <i>Leishmania (Viannia) panamensis</i> Infections.

Physiological magnesium concentrations increase fidelity of diverse reverse transcriptases from HIV-1, HIV-2, and foamy virus, but not MuLV or AMV

Draft Genome Sequence of Pseudomonas aeruginosa Strain PA14-UM

The transition of M-CSF–derived human macrophages to a growth-promoting phenotype

Gene expression network analyses during infection with virulent and avirulent Trypanosoma cruzi strains unveil a role for fibroblasts in neutrophil recruitment and activation

Identification of Zinc-Dependent Mechanisms Used by Group B Streptococcus To Overcome Calprotectin-Mediated Stress

Collaborators (5)

Thomas Kehl-Fie

Jeffrey DeStefano

Vincent T. Lee

Lindsey Burcham

Joanna Bridger

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