Nathaniel Snyder

Has grant

Associate Professor

Lewis Katz School of Medicine at Temple University

United States

Research Interests

Explore related searches

Professors in United States

Contact this professor

Get a free first email built on one of their papers, ready for you to edit and send yourself.

About

Nathaniel Snyder is an Associate Professor at Temple University Lewis Katz School of Medicine in the United States. His research areas include metabolic processes in cancer, histone modifications, and the implications of hormonal exposure in ovarian and glioblastoma contexts. Recent articles authored by Dr. Snyder explore themes such as histone deacetylation, metabolic regulation of cancer, and the effects of dietary components on liver health.

Recent Grants

Grant: Close

Quantification of compartmentalized metabolism in eukaryotic systems

Open Date: 2019-08-01

Close Date: 2024-04-30

Grant: Close

Metabolism of propionic acid

Open Date: 2017-09-01

Close Date: 2019-08-31

Grant: Close

The influence of prenatal maternal exposures on fetal sterol metabolomics

Open Date: 2016-09-15

Close Date: 2018-08-31

Grant: Close

Lipidomics of meconium in neurodevelopment

Open Date: 2016-04-22

Close Date: 2019-03-31

Grant: Close

Prenatal biomarkers of exposure and individual susceptibility to endocrine disrupting compounds

Open Date: 2016-02-01

Close Date: 2019-01-31

Articles (19)

Year:

2025

Year:

2025

Year:

2025

Year:

2025

Year:

2025

Year:

2024

Year:

2024

Year:

2024

Year:

2024

Year:

2024

Year:

2023

Year:

2023

Year:

2023

Year:

2023

Year:

2023

Year:

2022

Year:

2022

Year:

University of Pittsburgh School of Medicine

UNITED STATES

View details

ApplyKite TwinLearns as you apply

Make your next move.

Prepare your application

Review drafts before you send them.

Nathaniel Snyder

Research Interests

Explore related searches

Contact this professor

About

Recent Grants

Articles (19)

Class I histone deacetylases catalyze lysine lactylation

Metabolic Effects of Succinate Dehydrogenase Loss in Cancer

Recycling for a cleaner metabolism

KRAS G12V mutation-selective requirement for ACSS2 in colorectal adenoma formation.

Ovarian-Specific Cyp17A1 Overexpression in Female Mice: A Novel Model of Endogenous Testosterone Excess.

Lactate reprograms glioblastoma immunity through CBX3-regulated histone lactylation

Meconium as an Analyte for Androgen Exposure: Analysis Through Varying Maternal-Fetal Biomarkers.

Bempedoic acid suppresses diet-induced hepatic steatosis independently of ATP-citrate lyase.

Brown fat ATP-citrate lyase links carbohydrate availability to thermogenesis and guards against metabolic stress.

Acetate drives ovarian cancer quiescence via ACSS2-mediated acetyl-CoA production.

Itaconic acid underpins hepatocyte lipid metabolism in non-alcoholic fatty liver disease in male mice

Dynamic protein deacetylation is a limited carbon source for acetyl-CoA–dependent metabolism

Metabolic regulation of epigenetic drug resistance

Allosteric role of the citrate synthase homology domain of ATP citrate lyase

Aorta- and liver-generated TMAO enhances trained immunity for increased inflammation via ER stress/mitochondrial ROS/glycolysis pathways

The associations between prenatal phthalate exposure measured in child meconium and cognitive functioning of 12-month-old children in two cohorts at elevated risk for adverse neurodevelopment

Cumulus cell acetyl-CoA metabolism from acetate is associated with maternal age but only partially with oocyte maturity

Author Correction: mTORC2-AKT signaling to ATP-citrate lyase drives brown adipogenesis and de novo lipogenesis

mTORC2-AKT signaling to ATP-citrate lyase drives brown adipogenesis and de novo lipogenesis

Collaborators (5)

Daniel McVicar

George Burslem

John A. Haley

Emily Goldberg

Nadine Hempel

Make your next move.