Paula Moyano

Assistant Professor

Universidad Complutense de Madrid

Spain

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Paula Moyano is an Assistant Professor at the Complutense University of Madrid, Spain. Her research primarily focuses on neuroprotection, neurodegeneration, and the impact of environmental toxins on neural pathways. Recent publications include studies on the effects of thyroid hormones and various chemicals such as bisphenol A and cadmium on cholinergic and glutamatergic systems. Her work also explores the signaling pathways involved in cell death and neuroinflammation, emphasizing the development of selective kinase inhibitors.

Articles (12)

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2024

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2024

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2023

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2023

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2022

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2022

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2022

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2022

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2021

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2021

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2021

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2021

Collaborators (7)

José Carlos Plaza

Profesor Asociado

Universidad Complutense de Madrid

SPAIN

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Juan Francisco González

Profesor Titular de Universidad

Universidad Complutense de Madrid

SPAIN

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Kamil Paruch

CZECH REPUBLIC

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David Vicente-Zurdo

Assistant Professor

Universidad CEU San Pablo

SPAIN

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Javier Del Pino

Professor

Universidad Complutense de Madrid

SPAIN

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MARIA JOSE ANADON BASELGA

PROFESORA TITULAR

Universidad Complutense de Madrid

SPAIN

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Václav Němec

Goethe University Frankfurt

GERMANY

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Paula Moyano

Research Interests

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Articles (12)

Increased Levels of Phosphorylated-P38α Induce WNT/β-Catenin and NGF/P75NTR/TrkA Pathways Disruption and SN56 Cell Death following Single and Repeated Chlorpyrifos Treatment

Thyroid Hormone Neuroprotection Against Perfluorooctane Sulfonic Acid Cholinergic and Glutamatergic Disruption and Neurodegeneration Induction

Cadmium-promoted thyroid hormones disruption mediates ROS, inflammation, Aβ and Tau proteins production, gliosis, spongiosis and neurodegeneration in rat basal forebrain

Discovery of Potent and Exquisitely Selective Inhibitors of Kinase CK1 with Tunable Isoform Selectivity

Neuroprotective mechanisms of multitarget 7-aminophenanthridin-6(5H)-one derivatives against metal-induced amyloid proteins generation and aggregation

Insulin Signaling Disruption and INF-γ Upregulation Induce Aβ1–42 and Hyperphosphorylated-Tau Proteins Synthesis and Cell Death after Paraquat Treatment of Primary Hippocampal Cells

Single and repeated bisphenol A treatment induces ROS, Aβ and hyperphosphorylated-tau accumulation, and insulin pathways disruption, through HDAC2 and PTP1B overexpression, leading to SN56 cholinergic apoptotic cell death

Cadmium-induced neurotoxic effects on rat basal forebrain cholinergic system through thyroid hormones disruption

Neuroprotective Action of Multitarget 7-Aminophenanthridin-6(5H)-one Derivatives against Metal-Induced Cell Death and Oxidative Stress in SN56 Cells

Bisphenol A single and repeated treatment increases HDAC2, leading to cholinergic neurotransmission dysfunction and SN56 cholinergic apoptotic cell death through AChE variants overexpression and NGF/TrkA/P75NTR signaling disruption

Aryl Hydrocarbon Receptor Activation Produces Heat Shock Protein 90 and 70 Overexpression, Prostaglandin E2/Wnt/β-Catenin Signaling Disruption, and Cell Proliferation in MCF-7 and MDA-MB-231 Cells after 24 h and 14 Days of Chlorpyrifos Treatment

Chlorpyrifos induces cell proliferation in MCF-7 and MDA-MB-231 cells, through cholinergic and Wnt/β-catenin signaling disruption, AChE-R upregulation and oxidative stress generation after single and repeated treatment

Collaborators (7)

José Carlos Plaza

Juan Francisco González

Kamil Paruch

David Vicente-Zurdo

Javier Del Pino

MARIA JOSE ANADON BASELGA

Václav Němec

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