Rachael Philips

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National Institutes of Health

United States

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Consequences of a Stat1 gain-of-function mutation in gene expression and viral response

Open Date: 2020-09-01

Close Date: 2023-08-31

Articles (8)

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2025

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2024

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2023

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2023

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2022

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2021

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2021

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2020

Collaborators (6)

Yohei Mikami

Associate Professor

Keio University School of Medicine

JAPAN

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Geraldine Blanchard-Rohner

University Hospital of Geneva

SWITZERLAND

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Giuseppe Sciumè

Sapienza University of Rome

ITALY

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Yuka Kanno

National Institutes of Health

UNITED STATES

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Mana Momenilandi

Imagine Institute for Genetic Diseases

FRANCE

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Chen Yao

Assistant Professor

University of Texas Southwestern Medical Center

UNITED STATES

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Rachael Philips

Research Interests

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Contact this professor

Recent Grants

Articles (8)

An activating Stat1 mutant disrupts normal STAT4 innate lymphocyte programs during viral infection

A CTCF-binding site in the Mdm1-Il22-Ifng locus shapes cytokine expression profiles and plays a critical role in early Th1 cell fate specification.

Human germline heterozygous gain-of-function STAT6 variants cause severe allergic disease.

Variant <i>STAT4</i> and Response to Ruxolitinib in an Autoinflammatory Syndrome.

The JAK-STAT pathway at 30: Much learned, much more to do

MicroRNA-221 and -222 modulate intestinal inflammatory Th17 cell response as negative feedback regulators downstream of interleukin-23.

Histone deacetylase 3 represses cholesterol efflux during CD4<sup>+</sup> T-cell activation.

Translating JAKs to Jakinibs.

Collaborators (6)

Yohei Mikami

Geraldine Blanchard-Rohner

Giuseppe Sciumè

Yuka Kanno

Mana Momenilandi

Chen Yao

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