Rajesh Khanna

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Professor and Director, NYU Pain Research Center

New York University

United States

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Rajesh Khanna is a Professor and Director at the NYU Pain Research Center at New York University, USA. His research focuses on the molecular mechanisms of pain, specifically in relation to osteoarthritis and neuropathic pain. Recent articles authored by Professor Khanna explore topics such as NGF-TrkA signaling in chronic pain and the role of sodium channels and mast cell chymases in pain modulation.

Recent Grants

Grant: Close

CRMP2, Nav1.7 sodium channel, and chronic pain

Open Date: 2017-06-01

Close Date: 2022-02-28

Grant: Close

Regulators of NaV1.7 Channels: Novel Anti-Allodynic Drug Candidates

Open Date: 2017-05-01

Close Date: 2018-04-30

Grant: Close

CRMP2, mitochondria, and Huntington?s disease

Open Date: 2017-04-01

Close Date: 2022-02-28

Grant: Close

Drug Discovery for Treatment of pain and migraine in NF1

Open Date: 2016-01-04

Close Date: 2019-01-04

Grant: Close

Drug Discovery for Treatment of pain and migraine in NF1

Open Date: 2016-01-04

Close Date: 2019-01-04

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Collaborators (3)

A. A. Leslie Gunatilaka

UNITED STATES

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David Meyerholz

Professor

The University of Iowa Roy J and Lucille A Carver College of Medicine

UNITED STATES

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Dr Sascha Alles

Assistant Professor (Tenure-track)

University of New Mexico School of Medicine

UNITED STATES

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Rajesh Khanna

Research Interests

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About

Recent Grants

Articles (29)

Decoding chronic pain in OA via NGF-TrkA signaling

Beyond single targets: leveraging degeneracy in sodium channels for osteoarthritis analgesia

Mast cell chymases: essential for preventing the transition from acute to chronic pain

Ribosomes under stress: GCN2 integrates translational dysfunction and pain signaling

“N-credible” how a tiny viral protein is turning pain into a long-term gig

R and S enantiomers of CBD3063, a CaV2.2 N-type calcium channel modulator, alleviate capsaicin-induced inflammatory pain

Pain's puzzle pieces: MAGI-1, NaV1.8, degradation, analgesia

Collapsin Response Mediator Protein 2 (CRMP2) Modulates Mitochondrial Oxidative Metabolism in Knock-In AD Mouse Model

Unveiling the Dichotomic Nature of VEGFA in neuropathic pain: A path to targeted therapeutics

NF1+/ex42del miniswine model the cellular disruptions and behavioral presentations of NF1‐associated cognitive and motor impairment

Nociplastic pain mechanisms and toll-like receptors as promising targets for its management

SUMOylation and DeSUMOylation: Tug of War of Pain Signaling

Uncoupling the CRMP2-CaV2.2 Interaction Reduces Pain-Like Behavior in a Preclinical Joint-Pain Model

Effect of Crude Extract from the Sea Anemone Bunodeopsis globulifera on Voltage-Gated Ion Channels from Central and Peripheral Murine Nervous Systems

Biomarkers for Assessing Diet-Related Neurocognitive Deficits in Children—A Systematic Review

Small molecule targeting NaV1.7 via inhibition of CRMP2-Ubc9 interaction reduces pain-related outcomes in a rodent osteoarthritic model

Peptide and Peptidomimetic Inhibitors Targeting the Interaction of Collapsin Response Mediator Protein 2 with the N-Type Calcium Channel for Pain Relief

C–H Functionalization-Enabled 11-Step Semisynthesis of (−)-Veragranine A and Characterization of Synthetic Analogs in Osteoarthritis-related Pain Treatment

Regulating neuronal excitability: The role of S-palmitoylation in NaV1.7 activity and voltage sensitivity

Argentatin C Analogues with Potential Antinociceptive Activity and Other Triterpenoid Constituents from the Aerial Parts of Parthenium incanum

CRMP2 Participates in Regulating Mitochondrial Morphology and Motility in Alzheimer’s Disease

The Emerging Translational Potential of MNK Inhibitors for the Treatment of Chronic Pain

Plant and fungi derived analgesic natural products targeting voltage-gated sodium and calcium channels

Small molecule targeting NaV1.7 via inhibition of the CRMP2-Ubc9 interaction reduces pain in chronic constriction injury (CCI) rats

Some Like It Hot: Dynamic Control of Cav2.2 Channels By Chili Peppers

Neurodegenerative Diseases: From Dysproteostasis, Altered Calcium Signalosome to Selective Neuronal Vulnerability to AAV-Mediated Gene Therapy

Effect of morphine and ibuprofen on nociceptive behavior, preening and motor activity following tonic chemical pain in the Japanese quail (Coturnix japonica)

The small molecule compound C65780 alleviates pain by stabilizing voltage-gated sodium channels in the inactivated and slowly-recovering state

Neuronal allodynic mechanisms of Slc7a5 (LAT1) in the spared nerve injury rodent model of neuropathic pain

Collaborators (3)

A. A. Leslie Gunatilaka

David Meyerholz

Dr Sascha Alles

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