Rana Al-Sadi

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Penn State College of Medicine

United States

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MMP-9 Modulates Intestinal Epithelial Tight Junction Barrier and Intestinal Inflammation

Open Date: 2015-07-01

Close Date: 2019-02-28

Articles (13)

We present the complete genome sequence of the probiotic strain Lactobacillus acidophilus ATCC 9224. The genome sequence provides a valuable resource for investigating the phylogenetic evolution of this lineage and conducting comparative genomics with other Lactobacillus strains and species.

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Collaborators (4)

Manmeet Rawat

Assistant Professor

Penn State College of Medicine

UNITED STATES

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Prashant Nighot

Penn State College of Medicine

UNITED STATES

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Thomas Y. Ma

Penn State College of Medicine

UNITED STATES

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Arun Bhunia

Professor of Food Microbiology

Purdue University

UNITED STATES

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Rana Al-Sadi

Research Interests

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Contact this professor

Recent Grants

Articles (13)

The complete genome sequence of probiotic <i>Lactobacillus acidophilus</i> ATCC 9224 isolated from sour milk

Lactobacillus acidophilus inhibits the TNF-α-induced increase in intestinal epithelial tight junction permeability via a TLR-2 and PI3K-dependent inhibition of NF-κB activation

Editorial: Intestinal epithelial barrier disruption by enteric pathogens

Role of Bifidobacterium in Modulating the Intestinal Epithelial Tight Junction Barrier: Current Knowledge and Perspectives

Editorial: Role of matrix metalloproteinases and other inflammatory mediators in the disruption of the intestinal tight junction barrier

Lactobacillus acidophilus Induces a Strain-specific and Toll-Like Receptor 2–Dependent Enhancement of Intestinal Epithelial Tight Junction Barrier and Protection Against Intestinal Inflammation

<i>Bifidobacterium bifidum</i> Enhances the Intestinal Epithelial Tight Junction Barrier and Protects against Intestinal Inflammation by Targeting the Toll-like Receptor-2 Pathway in an NF-κB-Independent Manner.

Penicillin Allergy Label Increases Risk of Worse Clinical Outcomes in COVID-19.

Matrix Metalloproteinase-9 (MMP-9) induced disruption of intestinal epithelial tight junction barrier is mediated by NF-κB activation

IL-1β and the Intestinal Epithelial Tight Junction Barrier

IL1B Increases Intestinal Tight Junction Permeability by Up-regulation of MIR200C-3p, Which Degrades Occludin mRNA

Talk about micromanaging! Role of microRNAs in intestinal barrier function

Collaborators (4)

Manmeet Rawat

Prashant Nighot

Thomas Y. Ma

Arun Bhunia

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