Sarah Courtois

Associate Professor

Centre de Résonance Magnétique des Systèmes Biologiques

France

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Sarah Courtois is an Associate Professor at the Centre de Résonance Magnétique des Systèmes Biologiques in France. Her research focuses on pancreatic cancer, specifically the metabolic responses to microenvironmental cues, tumorigenicity, and the role of lipid metabolism in cancer stem cells. In addition, she investigates mitochondrial diseases and their neurodegenerative implications. Her recent publications highlight pharmacological strategies to target cancer metabolism and the influence of gut microbiota in disease progression.

Articles (10)

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2025

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2024

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2024

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2023

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2021

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2021

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2021

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2021

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2020

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2020

Collaborators (6)

Christian Frezza

Professor of metabolomics in ageing

University of Cologne

GERMANY

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VICTOR H. VILLAR

University of St. Andrews

UNITED KINGDOM

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Christopher Heeschen

Candiolo Cancer Institute

ITALY

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Christine Varon

Prof.

University of Bordeaux

FRANCE

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Piedad-del-Socorro Murdoch

Profesora titular

Universidad de Sevilla

SPAIN

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Raúl V. Durán

Universidad de Sevilla

SPAIN

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Sarah Courtois

Research Interests

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Articles (10)

PPARδ Orchestrates a Prometastatic Metabolic Response to Microenvironmental Cues in Pancreatic Cancer.

Fatty acid oxidation is critical for the tumorigenic potential and chemoresistance of pancreatic cancer stem cells

Mutation on MT-CO2 gene induces mitochondrial disease associated with neurodegeneration and intracerebral iron accumulation (NBIA)

Pharmacological targeting of the receptor ALK inhibits tumorigenicity and overcomes chemoresistance in pancreatic ductal adenocarcinoma

Inhibition of mitochondrial dynamics preferentially targets pancreatic cancer cells with enhanced tumorigenic and invasive potential

Two parallel pathways connect glutamine metabolism and mTORC1 activity to regulate glutamoptosis

Lipid droplets as metabolic determinants for stemness and chemoresistance in cancer.

Metformin Modifies the Gut Microbiota of Mice Infected with <i>Helicobacter pylori</i>.

Autophagy induced by Helicobacter pylori infection is necessary for gastric cancer stem cell emergence.

Molecular and Metabolic Subtypes Correspondence for Pancreatic Ductal Adenocarcinoma Classification.

Collaborators (6)

Christian Frezza

VICTOR H. VILLAR

Christopher Heeschen

Christine Varon

Piedad-del-Socorro Murdoch

Raúl V. Durán

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