Sarah Richardson

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Associate Professor in Cellular Biomedicine

University of Exeter

United Kingdom

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About

Sarah Richardson is an Associate Professor in Cellular Biomedicine at the University of Exeter, United Kingdom. Her research primarily focuses on the heterogeneity of type 1 diabetes, its pathogenesis, prevention, and treatment, as demonstrated in her recent publications. She also investigates the implications of pancreatic histology in diabetes and the role of enteroviruses in the disease process.

Recent Grants

Grant: Close

Phenotype and specificity of the islet inflammation in Type 1 diabetes

Open Date: 2018-06-01

Close Date: 2020-06-01

Grant: Close

The JDRF nPOD-Virus Group: Towards Vaccine Development

Open Date: 2017-11-01

Close Date: 2019-11-01

Grant: Close

Type 1 diabetes genetic risk and persistent beta cell enteroviral infection - a lethal combination?

Open Date: 2017-03-31

Close Date: 2020-03-30

Grant: Close

Type 1 diabetes genetic risk and persistent beta cell enteroviral infection - a lethal combination?

Open Date: 2017-03-31

Close Date: 2020-12-31

Grant: Close

Characterisation of immune cell interactions in the insulitic infiltrate of patients with recent-onset type 1 diabetes

Open Date: 2017-01-01

Close Date: 2019-12-31

Articles (22)

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2021

Collaborators (21)

Aaron Michels

Associate Professor

University of Colorado School of Medicine

UNITED STATES

Sarah Richardson

Research Interests

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About

Recent Grants

Articles (22)

Challenges and Opportunities for Understanding the Pathogenesis of Type 1 Diabetes: An Endocrine Society Scientific Statement

Quantitative analysis of human adult pancreatic histology reveals separate fatty and fibrotic phenotypes in type 2 diabetes

The Heterogeneity of Type 1 Diabetes: Implications for Pathogenesis, Prevention, and Treatment—2024 Diabetes, Diabetes Care, and Diabetologia Expert Forum

The Heterogeneity of Type 1 Diabetes: Implications for Pathogenesis, Prevention, and Treatment—2024 Diabetes, Diabetes Care, and Diabetologia Expert Forum

The heterogeneity of type 1 diabetes: implications for pathogenesis, prevention, and treatment—2024 Diabetes, Diabetes Care, and Diabetologia Expert Forum

Joint analysis of the nPOD-Virus Group data: the association of enterovirus with type 1 diabetes is supported by multiple markers of infection in pancreas tissue

Soluble HLA Class I Is Released From Human β-Cells Following Exposure to Interferons.

Enterovirus VP1 protein and HLA class I hyperexpression in pancreatic islet cells of organ donors with type 1 diabetes.

Type 1 Diabetes Genetic Risk Contributes to Phenotypic Presentation in Monogenic Autoimmune Diabetes.

Interferon-α promotes HLA-B-restricted presentation of conventional and alternative antigens in human pancreatic β-cells.

Regulation of STAT1 Signaling in Human Pancreatic β-Cells by the Lysine Deacetylase HDAC6: A New Therapeutic Opportunity in Type 1 Diabetes?

Mesenchymal stromal cells and their secretory products reduce the inflammatory crosstalk between islets and endothelial cells

Differential expression of genes controlling lymphocyte differentiation and migration in two distinct endotypes of type 1 diabetes

Milder loss of insulin-containing islets in individuals with type 1 diabetes and type 2 diabetes-associated TCF7L2 genetic variants

Images From the Exeter Archival Diabetes Biobank Now Accessible via Pancreatlas

Live enteroviruses, but not other viruses, detected in human pancreas at the onset of type 1 diabetes in the DiViD study

Detection of enterovirus RNA in peripheral blood mononuclear cells correlates with the presence of the predisposing allele of the type 1 diabetes risk gene IFIH1 and with disease stage

Circulating C-Peptide Levels in Living Children and Young People and Pancreatic β-Cell Loss in Pancreas Donors Across Type 1 Diabetes Disease Duration.

Temporal regulation of interferon signalling in human EndoC-βH1 cells.

Editorial: Immunopathology of Type 1 Diabetes.

Expression of CD47 in the pancreatic β-cells of people with recent-onset type 1 diabetes varies according to disease endotype.

Investigation of the utility of the 1.1B4 cell as a model human beta cell line for study of persistent enteroviral infection.

Collaborators (21)

Aaron Michels

Amir-Babak Sioofy-Khojine

Roberto Mallone

Riitta Veijola

DANIELA CAMPANI

Todd Brusko

Åke Lernmark

Julius O Nyalwidhe

Kyle Wedgwood

Stephen Rich

Michael R. Christie

Knut Dahl-Jorgensen

Dirk Homann

Mark Peakman

Noel G Morgan

Alberto Pugliese

sami oikarinen

Mark Turner

Gareth Hawkes

Maria J. Redondo

James Shaw

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