Shanti Diwakarla

co-Head Gut Barrier & Disease Laboratory

University of Melbourne

Australia

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Shanti Diwakarla is the co-Head of the Gut Barrier & Disease Laboratory at Melbourne University, Australia. Her research areas include gastrointestinal disorders, the impact of high-fat diets on immune functions, and the relationship between gut health and neurological diseases. Recent publications focus on the gastrointestinal function in Parkinson's disease, enterochromaffin cell analysis, and the implications of systemic inflammation on gut integrity.

Articles (16)

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2025

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2025

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2025

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2024

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2023

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2022

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2021

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2020

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2020

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2020

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2020

Collaborators (1)

Madeleine R. Di Natale

University of Melbourne

AUSTRALIA

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Shanti Diwakarla

Research Interests

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Articles (16)

Assessment of gastrointestinal function and enteric nervous system changes over time in the A53T mouse model of Parkinson’s disease

Acute exposure to high-fat diet impairs ILC3 functions and gut homeostasis

Stratification of enterochromaffin cells by single-cell expression analysis

Localisation of the relaxin-family peptide 3 receptor to enteroendocrine cells of the intestine in RXFP3-Cre/tdTomato mice

The role of the gastrointestinal barrier in obesity‐associated systemic inflammation

Leaky gut in systemic inflammation: exploring the link between gastrointestinal disorders and age-related diseases

Understanding the potential causes of gastrointestinal dysfunctions in multiple system atrophy

Gastrointestinal consequences of lipopolysaccharide-induced lung inflammation

A Critical Analysis of Intestinal Enteric Neuron Loss and Constipation in Parkinson’s Disease

Gastrointestinal Dysfunction in Parkinson’s Disease: Current and Potential Therapeutics

ATH434 Reverses Colorectal Dysfunction in the A53T Mouse Model of Parkinson’s Disease

The association of enteric neuropathy with gut phenotypes in acute and progressive models of Parkinson's disease.

Investigation of nerve pathways mediating colorectal dysfunction in Parkinson’s disease model produced by lesion of nigrostriatal dopaminergic neurons

Colokinetic effect of an insulin‐like peptide 5‐related agonist of the RXFP4 receptor

Insulin‐regulated aminopeptidase inhibitor‐mediated increases in dendritic spine density are facilitated by glucose uptake

Chronic isolation stress is associated with increased colonic and motor symptoms in the A53T mouse model of Parkinson’s disease

Collaborators (1)

Madeleine R. Di Natale

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