Sibylle Jablonka

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Senior lecturer, group leader

University Hospital Würzburg

Germany

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Dr. Sibylle Jablonka is a Senior Lecturer and Group Leader at University Hospital Würzburg, Germany. Her research focuses on spinal muscular atrophy, axonal dynamics, and therapeutic interventions for neurological disorders. Recent publications include studies on the mechanisms of neuronal models and cellular homeostasis, as well as the efficacy of various compounds in ameliorating motor neuron deficiencies.

Recent Grants

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Analyse von Krankheitsmechanismen bei IGHMBP2 Defizienz in humanen und murinen neuronalen und nicht-neuronalen Zellen

Open Date: 2022-04-01

Close Date: 2025-03-01

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Analysis of affected cellular targets causing cell-autonomous and non-cell autonomous disease mechanisms in a mouse model for spinal muscular atrophy with respiratory distress type 1 (SMARD1)

Open Date: 2022-01-01

Close Date: 2025-01-01

Grant: Close

Untersuchung von zellulären Krankheitsmechanismen, die zur spinalen Muskelatrophie mit Ateminsuffizienz Typ 1 (SMARD1) führen.

Open Date: 2021-01-01

Close Date: 2025-12-01

Grant: Close

Modulation of calcium channels in mouse models for spinal muscular atrophy (SMA).

Open Date: 2018-01-01

Close Date: 2021-01-01

Grant: Close

Analysis of Brain-Derived-Neurotrophic-Factor (BDNF) signaling in spinal muscular atrophy (SMA).

Open Date: 2017-01-01

Close Date: 2020-01-01

Sibylle Jablonka

Research Interests

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Recent Grants

Articles (15)

Munc13-1 restoration mitigates presynaptic pathology in spinal muscular atrophy

Impact of Polymorphic Microfibers for Establishment of Neuronal Model

RNA helicase IGHMBP2 regulates THO complex to ensure cellular mRNA homeostasis.

Disease Mechanisms and Therapeutic Approaches in SMARD1—Insights from Animal Models and Cell Models

The SMA Modifier Plastin 3 Targets Cell Membrane-Associated Proteins in Motoneurons

Nifedipine Ameliorates Cellular Differentiation Defects of Smn-Deficient Motor Neurons and Enhances Neuromuscular Transmission in SMA Mice

CDNF rescues motor neurons in models of amyotrophic lateral sclerosis by targeting endoplasmic reticulum stress

Plastin 3 rescues cell surface translocation and activation of TrkB in spinal muscular atrophy

Impaired dynamic interaction of axonal endoplasmic reticulum and ribosomes contributes to defective stimulus–response in spinal muscular atrophy

Therapy development for spinal muscular atrophy: perspectives for muscular dystrophies and neurodegenerative disorders.

Pegylated Insulin-Like Growth Factor 1 attenuates Hair Cell Loss and promotes Presynaptic Maintenance of Medial Olivocochlear Cholinergic Fibers in the Cochlea of the Progressive Motor Neuropathy Mouse

Interaction of 7SK with the Smn complex modulates snRNP production

Loss of full-length hnRNP R isoform impairs DNA damage response in motoneurons by inhibiting Yb1 recruitment to chromatin.

Dynamic remodeling of ribosomes and endoplasmic reticulum in axon terminals of motoneurons

Loss of Tdp-43 disrupts the axonal transcriptome of motoneurons accompanied by impaired axonal translation and mitochondria function

Collaborators (9)

Julieth Sierra-Delgado

Päivi Pulkkila

Michael Sendtner

Lucia Tabares

Markus Sauer

Michael Briese

Felix Meissner

Utz Fischer

Merja H Voutilainen

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