Stefano Santaguida

Associate Professor

University of Milan

Italy

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Dr. Stefano Santaguida is an Associate Professor at the University of Milan, Italy. His research primarily focuses on cell division, genome stability, and the biological implications of aneuploidy. Recent articles highlight his contributions to understanding the mechanisms of DNA damage response and protein degradation in human aneuploid cells, as well as the role of sphingolipid synthesis in maintaining cellular integrity.

Articles (14)

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2025

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2024

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2024

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2024

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2024

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2024

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2023

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2022

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2022

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2021

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2021

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2021

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2021

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2021

Collaborators (10)

Uri Ben-David

ISRAEL

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Takashi Hiiragi

Kyoto University

JAPAN

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Michael Mülleder

Head

Charité - Universitätsmedizin Berlin

GERMANY

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Heidi McBride

Professor

Montreal Neurological Institute and Hospital

CANADA

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Edridge D'Souza

Columbia University

UNITED STATES

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Peter Ly

Assistant Professor

University of Texas Southwestern Medical Center

UNITED STATES

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On Sun Lau

Assistant Professor

National University of Singapore (NUS)

SINGAPORE

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Lihua Zhu

Professor and Head of Bioinformatics Core

University of Massachusetts Chan Medical School

UNITED STATES

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Flavia Biamonte

Associate Professor

Università degli Studi Mediterranea di Reggio Calabria

ITALY

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Julia Muenzner

Charité - Universitätsmedizin Berlin

GERMANY

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Stefano Santaguida

Research Interests

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Articles (14)

Sphingolipid synthesis maintains nuclear membrane integrity and genome stability during cell division

A p62-dependent rheostat dictates micronuclei catastrophe and chromosome rearrangements.

Micronuclear collapse from oxidative damage.

Human aneuploid cells depend on the RAF/MEK/ERK pathway for overcoming increased DNA damage.

Increased RNA and protein degradation is required for counteracting transcriptional burden and proteotoxic stress in human aneuploid cells.

Two decades of chromosomal instability and aneuploidy.

Profilin 1 deficiency drives mitotic defects and reduces genome stability

Regulation of protein complex partners as a compensatory mechanism in aneuploid tumors

Generation of aneuploid cells and assessment of their ability to survive in presence of chemotherapeutic agents.

Crotonylation directs the spindle

20 years of Developmental Cell: Looking forward.

Biomedical omics: first insights of a new MSc degree of the University of Milan.

Aneuploid senescent cells activate NF‐κB to promote their immune clearance by NK cells

Gene copy-number changes and chromosomal instability induced by aneuploidy confer resistance to chemotherapy.

Collaborators (10)

Uri Ben-David

Takashi Hiiragi

Michael Mülleder

Heidi McBride

Edridge D'Souza

Peter Ly

On Sun Lau

Lihua Zhu

Flavia Biamonte

Julia Muenzner

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