Stephania Libreros

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Assistant Professor

Yale University

United States

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About

Dr. Stephania Libreros is an Assistant Professor at Yale University, USA. Her research focuses on the roles of nanoparticles and lipid metabolism in inflammation, cancer, and immune response. Recent articles by Dr. Libreros explore the mechanisms of neutrophil behavior, the impact of specialized pro-resolving mediators, and the relationship between immune profiles and emotional reactivity. She is dedicated to advancing the understanding of immune responses in various physiological and pathological contexts.

Recent Grants

Grant: Close

Bone Marrow Functions of Novel Pro-Resolving Mediators

Open Date: 2020-08-01

Close Date: 2022-07-31

Grant: Close

Novel Role of Pro-Resolving Lipid Mediators in Signaling Leukocytes

Open Date: 2018-06-01

Close Date: 2021-05-31

Articles (9)

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2025

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2024

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2024

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2024

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2023

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2023

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2023

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2021

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2020

Collaborators (3)

Charles N. Serhan

Professor and Center Director

Brigham and Women's Hospital and Harvard Medical School

UNITED STATES

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Nan Chiang

Assistant Professor

Brigham and Women's Hospital

UNITED STATES

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Philip Demokritou

Henry Rutgers Chair and Professor

Rutgers University

UNITED STATES

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Stephania Libreros

Research Interests

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About

Recent Grants

Articles (9)

SiO2 nanoparticles as disruptors of endogenous resolution mechanisms of inflammatory responses that exacerbate pneumonia

Is Lipid Metabolism of Value in Cancer Research and Treatment? Part I- Lipid Metabolism in Cancer

Is Lipid Metabolism of Value in Cancer Research and Treatment? Part II: Role of Specialized Pro-Resolving Mediators in Inflammation, Infections, and Cancer

The neonatal liver hosts a spontaneously occurring neutrophil population, exhibiting distinct spatial and functional characteristics from adults

Infectious neutrophil deployment is regulated by resolvin D4

Stereochemistry and functions of the new cysteinyl‐resolvin, 4S,5R‐RCTR1, in efferocytosis and erythrophagocytosis of human senescent erythrocytes

Author Correction: Human macrophages differentially produce specific resolvin or leukotriene signals that depend on bacterial pathogenicity

Cysteinyl-specialized proresolving mediators link resolution of infectious inflammation and tissue regeneration via TRAF3 activation

Inter-individual differences in immune profiles of outbred rats screened for an emotional reactivity phenotype.

Collaborators (3)

Charles N. Serhan

Nan Chiang

Philip Demokritou

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