Sue-Ann Mok

Assistant Professor

University of Alberta

Canada

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Dr. Sue-Ann Mok is an Assistant Professor at the University of Alberta, Canada. Her research focuses on Alzheimer's disease and related tauopathies, with recent publications exploring topics such as protein aggregation, microglial responses, and the role of gangliosides in extracellular vesicle secretion. Dr. Mok's work aims to advance the understanding of neurodegenerative mechanisms and potential therapeutic approaches in Alzheimer's pathology.

Articles (16)

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2025

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2024

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2024

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2023

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2023

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2023

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2022

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2022

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2021

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2021

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2021

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2020

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2020

Collaborators (16)

Jae-Young Cho

Adjunct Professor

University of Alberta

CANADA

Sue-Ann Mok

Research Interests

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Articles (16)

Gangliosides Modulate the Secretion of Extracellular Vesicles and Their Misfolded Protein Cargo

Inducers and modulators of protein aggregation in Alzheimer's disease - Critical tools for understanding the foundations of aggregate structures.

Native PLGA nanoparticles attenuate Aβ-seed induced tau aggregation under in vitro conditions: potential implication in Alzheimer’s disease pathology

Human iPSC 4R tauopathy model uncovers modifiers of tau propagation

Alzheimer's disease-linked risk alleles elevate microglial cGAS-associated senescence and neurodegeneration in a tauopathy model

Tau activation of microglial cGAS–IFN reduces MEF2C-mediated cognitive resilience

Disease-Associated Mutations in Tau Encode for Changes in Aggregate Structure Conformation

Erratum: SIRT1 Deacetylates Tau and Reduces Pathogenic Tau Spread in a Mouse Model of Tauopathy (The Journal of Neuroscience (2018) 11 (3680–3688) DOI: 10.1523/JNEUROSCI.2369-17.2018)

Streamlined high-throughput cloning protocol to generate arrayed mutant libraries

Tau interactome maps synaptic and mitochondrial processes associated with neurodegeneration

Microglial NF-κB drives tau spreading and toxicity in a mouse model of tauopathy

Functional genomics screen identifies proteostasis targets that modulate prion protein (PrP) stability.

Pathologic tau conformer ensembles induce dynamic, liquid-liquid phase separation events at the nuclear envelope.

Deciphering Network Crosstalk: The Current Status and Potential of miRNA Regulatory Networks on the HSP40 Molecular Chaperone Network.

Prion-Like Propagation Mechanisms in Tauopathies and Traumatic Brain Injury: Challenges and Prospects

Promoting tau secretion and propagation by hyperactive p300/CBP via autophagy-lysosomal pathway in tauopathy.

Collaborators (16)

Jae-Young Cho

Avi Samelson

Simonetta Sipione

Michael Ward

Tara Tracy

Evripidis Gavathiotis

Sang-Gyun Kang

Timothy Chang

Hugo McGurran

Rebecca Sereda

Isabelle Taylor

Ted Allison

Li Fan

Valerie Sim

Olivier Julien

ALICE MARIA GIANI

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