Taija Makinen

Professor

Uppsala University

Sweden

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About

Professor Taija Makinen is a faculty member at Uppsala University in Sweden. Her research focuses on various aspects of vascular biology, including lymphatic endothelial cells, venous malformations, and the dynamic regulation of the cytoskeleton. Recent publications highlight her work on angiopoietin signaling, genetic targeting for conditional genetics, and mechanisms of bone remodeling. Professor Makinen's expertise contributes significantly to the understanding of vascular and lymphatic systems.

Articles (23)

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2025

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2022

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2021

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2021

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2021

Collaborators (29)

Pia Ostergaard

Professor of Human Genetics

St George’s, University of London

UNITED KINGDOM

Taija Makinen

Research Interests

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About

Articles (23)

Dynamic cytoskeletal regulation of cell shape supports resilience of lymphatic endothelium

Angiopoietin–TIE2 feedforward circuit promotes PIK3CA-driven venous malformations

Direct specification of lymphatic endothelium from mesenchymal progenitors

Intravital Imaging of Disease Mechanisms in a Mouse Model of CCM Skin Lesions—Brief Report

iSuRe-HadCre is an essential tool for effective conditional genetics

Lymphatic malformations: mechanistic insights and evolving therapeutic frontiers

Specialized post-arterial capillaries facilitate adult bone remodelling

Genetic targeting of lymphatic endothelial cells in mice

APOLD1 loss causes endothelial dysfunction involving cell junctions, cytoskeletal architecture, and Weibel-Palade bodies, while disrupting hemostasis.

Hemogenic activity of lymphatic endothelium unleashed

Mapping the lymphatic system across body scales and expertise domains

Molecular anatomy of adult mouse leptomeninges

Penile cavernous sinusoids are Prox1-positive hybrid vessels

Immune-interacting lymphatic endothelial subtype at capillary terminals drives lymphatic malformation

Lymphangiogenesis requires Ang2/Tie/PI3K signaling for VEGFR3 cell-surface expression

The SARS-CoV-2 receptor ACE2 is expressed in mouse pericytes but not endothelial cells

Cdh5-lineage–independent origin of dermal lymphatics shown by temporally restricted lineage tracing

Cellular Origin of Sporadic CCMs.

Angiogenesis depends upon EPHB4-mediated export of collagen IV from vascular endothelial cells

Lymphangiogenesis requires Ang2/Tie/PI3K signaling for VEGFR3 cell-surface expression

Mutations in EPHB4 cause human venous valve aplasia.

An inducible Cldn11‐CreERT2 mouse line for selective targeting of lymphatic valves

Homeostatic maintenance of the lymphatic vasculature.

Collaborators (29)

Pia Ostergaard

Kari Vaahtomeri

Maria H Ulvmar

Miikka Vikkula

Per Nilsson

Alexander Gow

Katrine Dahl Bjørnholm

Andrey Anisimov

Mateusz Majda

Marie-christine ALESSI

Francesca Del Gaudio

Zorina Galis

Thom Saunders

Marie Jeansson

Marco Castro

Andrei Chagin

Pipsa Saharinen

Vishal Mohanakrishnan

eulalia baselga

Maarja Andaloussi Mäe

Hyun-Woo Jeong

Eric Thelin

Roland Fleck

Karthik Amudhala Hemanthakumar

Markus Otto Hjalmar Ramste

Jorge Andrade

Cord Brakebusch

Hiroo Suami

Maguelonne Roux

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