Taiki Abe

assistant professor

Tohoku University graduate school of medicine

Japan

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Dr. Taiki Abe is an assistant professor at Tohoku University School of Medicine in Japan. His research focuses on cellular senescence, lipid signaling, and genetic mutations related to various syndromes, as evidenced by his recent publications. Notable areas of study include the implications of LZTR1 mutations in disease phenotypes and the effects of lipid rafts on inflammatory responses. Dr. Abe's work contributes to understanding the molecular mechanisms underlying complex health conditions.

Articles (13)

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2025

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2024

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2024

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2023

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2023

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2022

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2022

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2021

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2020

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2020

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2020

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2020

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2020

Collaborators (5)

Yoshikazu Hayashi

Assistant professor

Fukuoka Dental College

JAPAN

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Kazuhiko Igarashi

Prof.

Tohoku University graduate school of medicine

JAPAN

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Ryo Takita

Professor

Shizuoka University

JAPAN

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Shin-ichi Inoue

University of Pennsylvania

UNITED STATES

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TAKUYA Noguchi

Associate professor

Tohoku University

JAPAN

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Taiki Abe

Research Interests

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Articles (13)

Elaidic acid drives cellular senescence and inflammation via lipid raft-mediated IL-1R signaling.

Lymphatic endothelial cell-specific <i>NRAS</i> p.Q61R mutant embryos show abnormal lymphatic vessel morphogenesis

Dysregulation of RAS proteostasis by autosomal-dominant LZTR1 mutation induces Noonan syndrome-like phenotypes in mice

Mecom mutation related to radioulnar synostosis with amegakaryocytic thrombocytopenia reduces HSPCs in mice

LZTR1 deficiency exerts high metastatic potential by enhancing sensitivity to EMT induction and controlling KLHL12-mediated collagen secretion

Phenotypic heterogeneity in individuals with MECOM variants in 2 families

Duplications in the G3 domain or switch II region in HRAS identified in patients with Costello syndrome

Chronic Hypercapnic Respiratory Failure in an Adult Patient with Silver-Russell Syndrome: A Case Report.

Antiepileptic Drug–Activated Constitutive Androstane Receptor Inhibits Peroxisome Proliferator–Activated Receptor α and Peroxisome Proliferator–Activated Receptor γ Coactivator 1α–Dependent Gene Expression to Increase Blood Triglyceride Levels

PXR Functionally Interacts with NF-κB and AP-1 to Downregulate the Inflammation-Induced Expression of Chemokine CXCL2 in Mice.

Costello syndrome model mice with a Hras G12S mutation are susceptible to develop house dust mite-induced atopic dermatitis

LZTR1 facilitates polyubiquitination and degradation of RAS-GTPases

Antiepileptic drug-activated constitutive androstane receptor inhibits peroxisome proliferator-activated receptor a and peroxisome proliferator-activated receptor g coactivator 1a-dependent gene expression to increase blood triglyceride levels

Collaborators (5)

Yoshikazu Hayashi

Kazuhiko Igarashi

Ryo Takita

Shin-ichi Inoue

TAKUYA Noguchi

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