TAKUYA Noguchi

Associate professor

Tohoku University

Japan

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Dr. Takuya Noguchi is an Associate Professor at Tohoku University in Japan. His research primarily focuses on cellular responses to oxidative stress, inflammation, and apoptosis, as evidenced by his recent works on the roles of various proteins and signaling pathways. He has contributed significantly to understanding the mechanisms underlying renal failure and inflammatory responses, particularly through the NLRP3 inflammasome and caspase pathways.

Articles (24)

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2025

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2021

Collaborators (9)

AKIRA NAGANUMA

Emeritus Professor

Tohoku University

JAPAN

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Guang Hong

Professor and Chair

Tohoku University

JAPAN

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Pascal Schneider

Senior researcher and lecturer

University of Lausanne

SWITZERLAND

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Gi-Wook Hwang

Tohoku Medical and Pharmaceutical University

JAPAN

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Tada-aki Kudo

Tohoku University

JAPAN

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Atsushi Matsuzawa

JAPAN

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Taiki Abe

assistant professor

Tohoku University graduate school of medicine

JAPAN

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Ryo Takita

Professor

Shizuoka University

JAPAN

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Hiroki Tsukamoto

Associate Professor

International University of Health and Welfare

JAPAN

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TAKUYA Noguchi

Research Interests

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About

Articles (24)

FBXO22 Suppresses Oxidative Stress-Induced ASK1 Activation and Cell Death via Ubiquitination-Dependent Degradation of TRIM48

Elaidic acid drives cellular senescence and inflammation via lipid raft-mediated IL-1R signaling.

The antirheumatic drug sulfasalazine ameliorates acute renal failure (ARF) induced by polymyxin B

Truncated LKB1 nonenzymatically enhances Fas-induced apoptosis by acting as a surrogate of Smac.

Frequency-Regulated Repeated Micro-Vibration Promotes Osteoblast Differentiation Through BMP Signaling in MC3T3-E1 Cells

The Selective 3-MST Inhibitor I3MT-3 Works as a Potent Caspase-1 Inhibitor

Benzalkonium chloride initiates proinflammatory responses via NLRP3 inflammasome activation

The degree of caspase-3 aggregation determines the selectivity of arsenic-induced cell death.

The NLRP3 Inflammasome Is a Major Cause of Acute Renal Failure Induced by Polypeptide Antibiotics

Roquin-2 promotes oxidative stress-induced cell death by ubiquitination-dependent degradation of TAK1

The E3 Ubiquitin Protein Ligase LINCR Amplifies the TLR-Mediated Signals through Direct Degradation of MKP1

Aggregability of the SQSTM1/p62-based aggresome-like induced structures determines the sensitivity to parthanatos

Physical Stimulation Methods Developed for In Vitro Neuronal Differentiation Studies of PC12 Cells: A Comprehensive Review

Methylmercury directly modifies the 105th cysteine residue in oncostatin M to promote binding to tumor necrosis factor receptor 3 and inhibit cell growth

Reactive sulfur species disaggregate the SQSTM1/p62-based aggresome-like induced structures via the HSP70 induction and prevent parthanatos

The NLRP3 Inflammasome Works as a Sensor for Detecting Hypoactivity of the Mitochondrial Src Family Kinases

LLPS of SQSTM1/p62 and NBR1 as outcomes of lysosomal stress response limits cancer cell metastasis

SP600125 Enhances Temperature-Controlled Repeated Thermal Stimulation-Induced Neurite Outgrowth in PC12-P1F1 Cells

The Distinct Roles of LKB1 and AMPK in p53-Dependent Apoptosis Induced by Cisplatin

The polypeptide antibiotic polymyxin B acts as a pro-inflammatory irritant by preferentially targeting macrophages

trans-Fatty acids promote p53-dependent apoptosis triggered by cisplatin-induced DNA interstrand crosslinks via the Nox-RIP1-ASK1-MAPK pathway

Mechanisms of gefitinib-induced interstitial pneumonitis: why and how the TKI perturbs innate immune systems?

The E3 Ubiquitin-Protein Ligase RNF4 Promotes TNF-α-Induced Cell Death Triggered by RIPK1

Methylmercury induces neuronal cell death by inducing TNF-α expression through the ASK1/p38 signaling pathway in microglia.

Collaborators (9)

AKIRA NAGANUMA

Guang Hong

Pascal Schneider

Gi-Wook Hwang

Tada-aki Kudo

Atsushi Matsuzawa

Taiki Abe

Ryo Takita

Hiroki Tsukamoto

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