Tommy Lewis

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Assistant Member

Oklahoma Medical Research Foundation

United States

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About

Dr. Tommy Lewis is an Assistant Member at the Oklahoma Medical Research Foundation in the United States. His research focuses on mitochondrial dynamics, neurodegeneration, and the cellular mechanisms underlying neuron function and development. Recent publications include studies on P-glycoprotein's effects on mitochondrial measurements in T cells, as well as the role of exercise-induced mitochondrial remodeling in preventing age-related neurodegeneration.

Recent Grants

Grant: Close

Molecular and cellular mechanisms regulating mitochondrial subpopulation dynamics and function in vivo

Open Date: 2020-08-01

Close Date: 2025-05-31

Grant: Close

In vivo investigation of mitochondrial dynamics in the mouse brain

Open Date: 2015-07-15

Close Date: 2017-06-30

Grant: Close

LKB1 function in axon development through regulation of mitochondrial trafficking

Open Date: 2012-07-01

Close Date: 2015-06-30

Articles (12)

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2025

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2025

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2025

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2024

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2023

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2023

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2022

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2022

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2021

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2021

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2021

Collaborators (6)

Francesco Petrelli

University of Lausanne

SWITZERLAND

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Benoit Viollet

INSERM

FRANCE

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Mirko Santello

University of Zurich

SWITZERLAND

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Franck Polleux

Professor

Columbia University

UNITED STATES

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Jacob Brown

Florida State University

UNITED STATES

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Benjamin Miller

Department Chair and Professor

Oklahoma Medical Research Foundation

UNITED STATES

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Tommy Lewis

Research Interests

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About

Recent Grants

Articles (12)

P-glycoprotein expression skews mitochondrial dye measurements in T cells.

Combining In Vivo 2-Photon Imaging with Photoactivatable Fluorescent Labeling Shows Low Rates of Mitochondrial Dynamics in Skeletal Muscle.

Siah2 antagonism of Pard3/JamC modulates Ntn1-Dcc signaling to regulate cerebellar granule neuron germinal zone exit

Metformin treatment results in distinctive skeletal muscle mitochondrial remodeling in rats with different intrinsic aerobic capacities.

Activity-dependent compartmentalization of dendritic mitochondria morphology through local regulation of fusion-fission balance in neurons in vivo.

Exercise and mitochondrial remodeling to prevent age-related neurodegeneration

Neuronal deletion of MnSOD in mice leads to demyelination, inflammation and progressive paralysis that mimics phenotypes associated with progressive multiple sclerosis

Aβ42 oligomers trigger synaptic loss through CAMKK2-AMPK-dependent effectors coordinating mitochondrial fission and mitophagy

Neuronal mitochondrial morphology is significantly affected by both fixative and oxygen level during perfusion

Mitochondrial biogenesis in developing astrocytes regulates astrocyte maturation and synapse formation

Axon morphogenesis and maintenance require an evolutionary conserved safeguard function of Wnk kinases antagonizing Sarm and Axed

Necroptosis increases with age in the brain and contributes to age-related neuroinflammation

Collaborators (6)

Francesco Petrelli

Benoit Viollet

Mirko Santello

Franck Polleux

Jacob Brown

Benjamin Miller

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